Calponin phosphorylation in cerebral cortex microvessels mediates sustained vasoconstriction after brain trauma. (Q48107590)

8 February 2018

title

Calponin phosphorylation in cerebral cortex microvessels mediates sustained vasoconstriction after brain trauma. (English)

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8 February 2018

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Christian W Kreipke

1

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8 February 2018

Randy Morgan

2

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8 February 2018

George Roberts

3

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8 February 2018

Mihir Bagchi

4

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8 February 2018

José A Rafols

5

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8 February 2018

publication date

1 June 2007

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8 February 2018

published in

Neurological Research

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8 February 2018

volume

29

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8 February 2018

issue

4

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8 February 2018

page(s)

369-374

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8 February 2018

Involvement of calponin and caldesmon in sustained contraction of arterial smooth muscle

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Calponin and caldesmon cellular domains in reacting microvessels following traumatic brain injury.

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Subcellular redistribution of calponin underlies sustained vascular contractility following traumatic brain injury.

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Upregulation of iNOS expression and phosphorylation of eIF-2alpha are paralleled by suppression of protein synthesis in rat hypothalamus in a closed head trauma model

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Fallen arches, or how the vertebrate got its head

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Spatial alterations in endothelin receptor expression are temporally associated with the altered microcirculation after brain trauma.

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Acute alterations of endothelin-1 and iNOS expression and control of the brain microcirculation after head trauma

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21 January 2018

Identifiers

DOI

10.1179/016164107X204684

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8 February 2018

PubMed ID

17626732

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8 February 2018