The gastric H+,K+-ATPase is a major autoantigen in chronic Helicobacter pylori gastritis with body mucosa atrophy. (Q47787226)

5 February 2018

title

The gastric H+,K+-ATPase is a major autoantigen in chronic Helicobacter pylori gastritis with body mucosa atrophy. (English)

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5 February 2018

Helicobacter pylori gastritis

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Claeys D

1

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5 February 2018

Faller G

2

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5 February 2018

Appelmelk BJ

3

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5 February 2018

Negrini R

4

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5 February 2018

Kirchner T

5

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5 February 2018

language of work or name

English

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publication date

1 August 1998

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5 February 2018

published in

Gastroenterology

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5 February 2018

volume

115

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5 February 2018

issue

2

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5 February 2018

page(s)

340-347

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5 February 2018

Helicobacter pylori infection in peptic ulcer disease

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Antigastric autoantibodies in Helicobacter pylori gastritis: prevalence, in-situ binding sites and clues for clinical relevance

7 January 2021

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Antigenic mimicry between Helicobacter pylori and gastric mucosa in the pathogenesis of body atrophic gastritis

7 January 2021

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Antigastric autoantibodies in Helicobacter pylori infection: implications of histological and clinical parameters of gastritis

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Apoptosis in chronic gastritis and its correlation with antigastric autoantibodies.

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Lipopolysaccharide of the Helicobacter pylori type strain NCTC 11637 (ATCC 43504): structure of the O antigen chain and core oligosaccharide regions

7 January 2021

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Lipopolysaccharides of Helicobacter pylori strains P466 and MO19: structures of the O antigen and core oligosaccharide regions

7 January 2021

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Classification and grading of gastritis. The updated Sydney System. International Workshop on the Histopathology of Gastritis, Houston 1994

7 January 2021

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Rapid purification of the gastric H+/K+-ATPase complex by tomato-lectin affinity chromatography

7 January 2021

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Primary structure and functional expression of the mouse and frog alpha-subunit of the gastric H(+)-K(+)-ATPase

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Neonatal injection of native proton pump antigens induces autoimmune gastritis in mice.

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Major parietal cell antigen in autoimmune gastritis with pernicious anemia is the acid-producing H+,K+-adenosine triphosphatase of the stomach

7 January 2021

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7 January 2021

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Positive correlation between H,K-adenosine triphosphatase autoantibodies and Helicobacter pylori antibodies in patients with pernicious anemia

7 January 2021

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Molecular mimicry between Helicobacter pylori and the host

7 January 2021

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Helicobacter pylori Lewis expression is related to the host Lewis phenotype

7 January 2021

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7 January 2021

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Parietal cell antibodies in pernicious anemia inhibit H+, K+-adenosine triphosphatase, the proton pump of the stomach.

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Antigastric autoantibodies and gastric secretory function in Helicobacter pylori-infected patients with duodenal ulcer and non-ulcer dyspepsia

7 January 2021

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Long-term sequelae of Helicobacter pylori gastritis.

7 January 2021

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The risk of stomach cancer in patients with gastric or duodenal ulcer disease

7 January 2021

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10.1016/S0016-5085(98)70200-8

7 January 2021

Identifiers

DOI

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5 February 2018

PubMed ID

9679039

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5 February 2018