Tumour necrosis factor-alpha released by testicular macrophages induces apoptosis of germ cells in autoimmune orchitis. (Q47613726)

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9 January 2020

title

Tumour necrosis factor-alpha released by testicular macrophages induces apoptosis of germ cells in autoimmune orchitis (English)

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9 January 2020

0 references

1

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9 January 2020

author name string

C Rival

2

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9 January 2020

S Jarazo-Dietrich

3

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9 January 2020

P Jacobo

4

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9 January 2020

V A Guazzone

5

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9 January 2020

L Lustig

6

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9 January 2020

language of work or name

English

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publication date

25 June 2008

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9 January 2020

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9 January 2020

volume

23

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9 January 2020

issue

8

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9 January 2020

page(s)

1865-1872

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Tumor necrosis factor-alpha promotes macrophage-induced vascular smooth muscle cell apoptosis by direct and autocrine mechanisms

9 January 2020

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Sequential study of the histopathology and cellular and humoral immune response during the development of an autoimmune orchitis in Wistar rats

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The response of testicular leukocytes to lipopolysaccharide-induced inflammation: further evidence for heterogeneity of the testicular macrophage population

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Estrogens up-regulate the Fas/FasL apoptotic pathway in lactotropes

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Tumor necrosis factor {alpha} reversibly disrupts the blood-testis barrier and impairs Sertoli-germ cell adhesion in the seminiferous epithelium of adult rat testes.

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Transgenic mice expressing p450 aromatase as a model for male infertility associated with chronic inflammation in the testis

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Nitric oxide mediates apoptosis through formation of peroxynitrite and Fas/Fas-ligand interactions in experimental autoimmune uveitis

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TNFalpha down-regulates the Fas ligand and inhibits germ cell apoptosis in the human testis.

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Microglial cell activation and proliferation precedes the onset of CNS autoimmunity

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TNF-alpha and IFN-gamma regulate expression and function of the Fas system in the seminiferous epithelium.

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Interleukin-6 and IL-6 receptor cell expression in testis of rats with autoimmune orchitis.

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An analysis of the role of tumor necrosis factor in the phenotypic expression of actively induced experimental allergic orchitis and experimental allergic encephalomyelitis

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