Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction. (Q47610191)

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English	Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction.	scientific article

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scholarly article

1 reference

Europe PubMed Central

4 February 2018

http://europepmc.org/abstract/MED/29030341

Macrophage overexpression of matrix metalloproteinase-9 in aged mice improves diastolic physiology and cardiac wound healing after myocardial infarction. (English)

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Europe PubMed Central

4 February 2018

http://europepmc.org/abstract/MED/29030341

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myocardial infarction

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Merry L. Lindsey

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Merry L Lindsey

7

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Andriy Yabluchanskiy

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Andriy Yabluchanskiy

4

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author name string

Cesar A Meschiari

1

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4 February 2018

http://europepmc.org/abstract/MED/29030341

Mira Jung

2

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Europe PubMed Central

4 February 2018

http://europepmc.org/abstract/MED/29030341

Rugmani Padmanabhan Iyer

3

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Europe PubMed Central

4 February 2018

http://europepmc.org/abstract/MED/29030341

Hiroe Toba

5

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Europe PubMed Central

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http://europepmc.org/abstract/MED/29030341

Michael R Garrett

6

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http://europepmc.org/abstract/MED/29030341

publication date

13 October 2017

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Europe PubMed Central

4 February 2018

http://europepmc.org/abstract/MED/29030341

American Journal of Physiology Heart and Circulatory Physiology

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4 February 2018

http://europepmc.org/abstract/MED/29030341

314

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H224-H235

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2

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4 February 2018

http://europepmc.org/abstract/MED/29030341

describes a project that uses

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Europe PubMed Central

11 June 2022

https://www.ebi.ac.uk/europepmc/webservices/rest/PMC5867652/fullTextXML

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Periodontal-induced chronic inflammation triggers macrophage secretion of Ccl12 to inhibit fibroblast-mediated cardiac wound healing

1 reference

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Detrimental role of lysyl oxidase in cardiac remodeling.

1 reference

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Cardiac Fibroblast Activation Post-Myocardial Infarction: Current Knowledge Gaps

1 reference

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29 September 2018

The impact of aging on cardiac extracellular matrix.

1 reference

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29 September 2018

Macrophage activation and polarization in post-infarction cardiac remodeling

1 reference

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29 September 2018

Transgenic overexpression of macrophage matrix metalloproteinase-9 exacerbates age-related cardiac hypertrophy, vessel rarefaction, inflammation, and fibrosis.

1 reference

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Early matrix metalloproteinase-9 inhibition post-myocardial infarction worsens cardiac dysfunction by delaying inflammation resolution

1 reference

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Defining the sham environment for post-myocardial infarction studies in mice

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MMP-9 signaling in the left ventricle following myocardial infarction

1 reference

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Temporal neutrophil polarization following myocardial infarction

1 reference

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29 September 2018

Matrix metalloproteinases as input and output signals for post-myocardial infarction remodeling

1 reference

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1 reference

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Cardioprotective effects of lysyl oxidase inhibition against volume overload-induced extracellular matrix remodeling

1 reference

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CD36 Is a Matrix Metalloproteinase-9 Substrate That Stimulates Neutrophil Apoptosis and Removal During Cardiac Remodeling.

1 reference

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A Novel Collagen Matricryptin Reduces Left Ventricular Dilation Post-Myocardial Infarction by Promoting Scar Formation and Angiogenesis

1 reference

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Matrix cross-linking lysyl oxidases are induced in response to myocardial infarction and promote cardiac dysfunction.

1 reference

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Building a better infarct: Modulation of collagen cross-linking to increase infarct stiffness and reduce left ventricular dilation post-myocardial infarction.

1 reference

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Deriving a cardiac ageing signature to reveal MMP-9-dependent inflammatory signalling in senescence.

1 reference

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Myocardial Infarction Superimposed on Aging: MMP-9 Deletion Promotes M2 Macrophage Polarization.

1 reference

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29 September 2018

Early matrix metalloproteinase-12 inhibition worsens post-myocardial infarction cardiac dysfunction by delaying inflammation resolution

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=5867652

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Heart Disease and Stroke Statistics--2015 Update: A Report From the American Heart Association

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P. gingivalis lipopolysaccharide intensifies inflammation post-myocardial infarction through matrix metalloproteinase-9

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Age-related changes in tissue macrophages precede cardiac functional impairment

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Cardiac wound healing post-myocardial infarction: a novel method to target extracellular matrix remodeling in the left ventricle

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Biochemistry and molecular biology of gelatinase B or matrix metalloproteinase-9 (MMP-9): the next decade

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Matrix metalloproteinase-9 deletion attenuates myocardial fibrosis and diastolic dysfunction in ageing mice

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Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction.

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Multi-analyte profiling reveals matrix metalloproteinase-9 and monocyte chemotactic protein-1 as plasma biomarkers of cardiac aging.

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Invited Review: Aging and the cardiovascular system

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Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction

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Role of platelet-derived growth factor in wound healing

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29 September 2018

Specific repair by discerning macrophages

1 reference

https://pubmed.ncbi.nlm.nih.gov/29030341

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Inhibition of plasminogen activators or matrix metalloproteinases prevents cardiac rupture but impairs therapeutic angiogenesis and causes cardiac failure

1 reference

https://pubmed.ncbi.nlm.nih.gov/29030341

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Specific temporal profile of matrix metalloproteinase release occurs in patients after myocardial infarction: relation to left ventricular remodeling

1 reference

https://pubmed.ncbi.nlm.nih.gov/29030341

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Overexpression of MMP9 in macrophages attenuates pulmonary fibrosis induced by bleomycin

1 reference

https://pubmed.ncbi.nlm.nih.gov/29030341

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1152/AJPHEART.00453.2017

1 reference

Europe PubMed Central

4 February 2018

http://europepmc.org/abstract/MED/29030341

0 references

1 reference

Europe PubMed Central

4 February 2018

http://europepmc.org/abstract/MED/29030341

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