Decreased blood-brain leptin transfer in an ovine model of obesity and weight loss: resolving the cause of leptin resistance (Q47446156)

16 January 2018

title

Decreased blood-brain leptin transfer in an ovine model of obesity and weight loss: resolving the cause of leptin resistance (English)

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16 January 2018

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C L Adam

1

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16 January 2018

P A Findlay

series ordinal

2

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16 January 2018

language of work or name

English

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publication date

9 February 2010

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International Journal of Obesity

published in

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16 January 2018

volume

34

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issue

6

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page(s)

980-988

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https://scigraph.springernature.com/pub.10.1038/ijo.2010.28

exact match

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cites work

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Molecular mechanisms associated with leptin resistance: n-3 polyunsaturated fatty acids induce alterations in the tight junction of the brain

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Hypothalamic gene expression in sheep for cocaine- and amphetamine-regulated transcript, pro-opiomelanocortin, neuropeptide Y, agouti-related peptide and leptin receptor and responses to negative energy balance

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Recombinant leptin for weight loss in obese and lean adults: a randomized, controlled, dose-escalation trial

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Tat-modified leptin is more accessible to hypothalamus through brain-blood barrier with a significant inhibition of body-weight gain in high-fat-diet fed mice

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Triglycerides induce leptin resistance at the blood-brain barrier

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Circulating leptin has saturable transport into intrathecal space in humans.

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Central leptin insufficiency syndrome: an interactive etiology for obesity, metabolic and neural diseases and for designing new therapeutic interventions

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Identifiers

DOI

10.1038/IJO.2010.28

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16 January 2018

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16 January 2018