Null mutations in Drosophila N-acetylglucosaminyltransferase I produce defects in locomotion and a reduced life span (Q47072658)

5 January 2018

title

Null mutations in Drosophila N-acetylglucosaminyltransferase I produce defects in locomotion and a reduced life span (English)

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5 January 2018

Mannosyl (alpha-1,3-)-glycoprotein beta-1,2-N-acetylglucosaminyltransferase Dmel_CG13431

main subject

Drosophila

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GOA release 2020-03-11

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Mohan Sarkar

1

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5 January 2018

Peter A Leventis

2

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5 January 2018

Cristina I Silvescu

3

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5 January 2018

Vernon N Reinhold

4

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Harry Schachter

5

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Gabrielle L Boulianne

6

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language of work or name

English

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publication date

7 March 2006

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5 January 2018

Journal of Biological Chemistry

published in

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volume

281

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issue

18

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5 January 2018

page(s)

12776-12785

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Post-translational modification of proteins and the discovery of new medicine

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Synthesis of paucimannose N-glycans by Caenorhabditis elegans requires prior actions of UDP-N-acetyl-D-glucosamine:alpha-3-D-mannoside beta1,2-N-acetylglucosaminyltransferase I, alpha3,6-mannosidase II and a specific membrane-bound beta-N-acetylgluc

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Five Lec1 CHO cell mutants have distinct Mgat1 gene mutations that encode truncated N-acetylglucosaminyltransferase I.

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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Insect cells encode a class II alpha-mannosidase with unique properties

21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.M512769200

1 reference

5 January 2018

PubMed ID

16522637

1 reference

5 January 2018