Molecular mechanism of imidapril for cardiovascular protection via inhibition of MMP-9. (Q46990084)

27 December 2017

title

Molecular mechanism of imidapril for cardiovascular protection via inhibition of MMP-9. (English)

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27 December 2017

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Daisuke Yamamoto

1

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27 December 2017

Shinji Takai

2

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27 December 2017

Denan Jin

3

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27 December 2017

Sachiko Inagaki

4

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27 December 2017

Kazuhiko Tanaka

5

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27 December 2017

Mizuo Miyazaki

6

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27 December 2017

publication date

16 August 2007

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27 December 2017

Journal of Molecular and Cellular Cardiology

published in

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27 December 2017

volume

43

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27 December 2017

issue

6

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27 December 2017

page(s)

670-676

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27 December 2017

Peripheral blood levels of matrix metalloproteases-2 and -9 are elevated in patients with acute coronary syndromes.

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Selective matrix metalloproteinase inhibition reduces left ventricular remodeling but does not inhibit angiogenesis after myocardial infarction.

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Matrix metalloproteinase-9 gene deletion facilitates angiogenesis after myocardial infarction.

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Captopril inhibits the 72 kDa and 92 kDa matrix metalloproteinases

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Cardiac remodelling in end stage heart failure: upregulation of matrix metalloproteinase (MMP) irrespective of the underlying disease, and evidence for a direct inhibitory effect of ACE inhibitors on MMP.

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A monoclonal antibody inhibits gelatinase B/MMP-9 by selective binding to part of the catalytic domain and not to the fibronectin or zinc binding domains

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Effects of zofenopril on myocardial ischemia in post-myocardial infarction patients with preserved left ventricular function: the Survival of Myocardial Infarction Long-term Evaluation (SMILE)-ISCHEMIA study

7 January 2021

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10.1016/J.YJMCC.2007.08.002

7 January 2021

Identifiers

DOI

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27 December 2017

PubMed ID

17884087

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27 December 2017