SHP2-mediated signaling cascade through gp130 is essential for LIF-dependent I CaL, [Ca2+]i transient, and APD increase in cardiomyocytes (Q46931098)

26 December 2017

title

SHP2-mediated signaling cascade through gp130 is essential for LIF-dependent I CaL, [Ca2+]i transient, and APD increase in cardiomyocytes (English)

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26 December 2017

author name string

Yoko Hagiwara

1

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26 December 2017

Shunichiro Miyoshi

2

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26 December 2017

Keiichi Fukuda

3

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26 December 2017

Nobuhiro Nishiyama

4

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26 December 2017

Yukinori Ikegami

5

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26 December 2017

Kojiro Tanimoto

6

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26 December 2017

Mitsushige Murata

7

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26 December 2017

Eiichi Takahashi

8

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26 December 2017

Kouji Shimoda

9

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26 December 2017

Toshio Hirano

10

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26 December 2017

Hideo Mitamura

11

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26 December 2017

Satoshi Ogawa

12

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26 December 2017

publication date

19 September 2007

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26 December 2017

Journal of Molecular and Cellular Cardiology

published in

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26 December 2017

volume

43

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26 December 2017

page(s)

710-716

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26 December 2017

issue

6

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26 December 2017

Leukemia inhibitory factor--a puzzling polyfunctional regulator

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Differentiation and growth arrest signals are generated through the cytoplasmic region of gp130 that is essential for Stat3 activation

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Leukemia inhibitory factor, a potent cardiac hypertrophic cytokine, activates the JAK/STAT pathway in rat cardiomyocytes

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Leukemia inhibitory factor, a potent cardiac hypertrophic cytokine, enhances L-type Ca2+ current and [Ca2+]i transient in cardiomyocytes

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Leukemia inhibitory factor activates cardiac L-Type Ca2+ channels via phosphorylation of serine 1829 in the rabbit Cav1.2 subunit

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Regulation of CCAAT/Enhancer binding protein, interleukin-6, interleukin-6 receptor, and gp130 expression during myocardial ischemia/reperfusion

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7 January 2021

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Complications associated with rapid caffeine application to cardiac myocytes that are not voltage clamped

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7 January 2021

Identifiers

DOI

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26 December 2017

PubMed ID

17961593

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26 December 2017