c-FLIP inhibits chemotherapy-induced colorectal cancer cell death (Q46772732)

25 December 2017

title

c-FLIP inhibits chemotherapy-induced colorectal cancer cell death (English)

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25 December 2017

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25 December 2017

author name string

D B Longley

1

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25 December 2017

T R Wilson

2

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25 December 2017

M McEwan

3

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25 December 2017

W L Allen

4

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25 December 2017

L Galligan

6

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P G Johnston

7

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25 December 2017

language of work or name

English

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publication date

1 February 2006

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25 December 2017

published in

Oncogene

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25 December 2017

volume

25

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25 December 2017

page(s)

838-848

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issue

6

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25 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1209122

exact match

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cites work

Oxaliplatin, fluorouracil, and leucovorin as adjuvant treatment for colon cancer.

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7 January 2021

Ultraviolet light induces apoptosis via direct activation of CD95 (Fas/APO-1) independently of its ligand CD95L

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Herpes simplex virus thymidine kinase/ganciclovir-induced apoptosis involves ligand-independent death receptor aggregation and activation of caspases.

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Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

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A domain in TNF receptors that mediates ligand-independent receptor assembly and signaling

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FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

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Quantitative analysis of dose-effect relationships: the combined effects of multiple drugs or enzyme inhibitors

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IL-4 protects tumor cells from anti-CD95 and chemotherapeutic agents via up-regulation of antiapoptotic proteins

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Irinotecan combined with fluorouracil compared with fluorouracil alone as first-line treatment for metastatic colorectal cancer: a multicentre randomised trial

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Phase III multicenter randomized trial of oxaliplatin added to chronomodulated fluorouracil-leucovorin as first-line treatment of metastatic colorectal cancer

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Depletion of membrane cholesterol causes ligand-independent activation of Fas and apoptosis

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TRAIL: a molecule with multiple receptors and control mechanisms

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Recruitment of TRADD, FADD, and caspase 8 to double-stranded RNA-triggered death inducing signaling complexes (dsRNA-DISCs).

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Inhibition of death receptor signals by cellular FLIP

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Fas-associated protein with death domain (FADD)-independent recruitment of c-FLIPL to death receptor 5.

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Up-regulation of FLIP in cisplatin-selected HeLa cells causes cross-resistance to CD95/Fas death signalling

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N-terminal fragment of c-FLIP(L) processed by caspase 8 specifically interacts with TRAF2 and induces activation of the NF-kappaB signaling pathway

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Transforming growth factor-beta 1 induces apoptosis through Fas ligand-independent activation of the Fas death pathway in human gastric SNU-620 carcinoma cells.

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FLICE-inhibitory proteins: regulators of death receptor-mediated apoptosis

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5-Fluorouracil: mechanisms of action and clinical strategies

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c-FLIP mediates resistance of Hodgkin/Reed-Sternberg cells to death receptor-induced apoptosis

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Role of MRIT/cFLIP in protection against chemotherapy-induced apoptosis

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Fas ligand-independent, FADD-mediated activation of the Fas death pathway by anticancer drugs.

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The long form of FLIP is an activator of caspase-8 at the Fas death-inducing signaling complex

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Rapid colorimetric assay for cellular growth and survival: Application to proliferation and cytotoxicity assays

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Fas ligand-induced apoptosis

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7 January 2021

Upregulation of FLIP(S) by Akt, a possible inhibition mechanism of TRAIL-induced apoptosis in human gastric cancers

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7 January 2021

Alternative splicing variants of c-FLIP transduce the differential signal through the Raf or TRAF2 in TNF-induced cell proliferation

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Regulation of Apo2L/tumor necrosis factor-related apoptosis-inducing ligand-induced apoptosis in thyroid carcinoma cells

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Increased expression of cFLIP(L) in colonic adenocarcinoma

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Intracellular regulation of Fas-induced apoptosis in human fibroblasts by extracellular factors and cycloheximide

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The role of c-FLIP in modulation of CD95-induced apoptosis

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Inducible silencing of KILLER/DR5 in vivo promotes bioluminescent colon tumor xenograft growth and confers resistance to chemotherapeutic agent 5-fluorouracil

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Requirement for Casper (c-FLIP) in regulation of death receptor-induced apoptosis and embryonic development

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7 January 2021

Overexpression of cellular FLICE-inhibitory protein (FLIP) in gastric adenocarcinoma.

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1209122

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25 December 2017

Dimensions Publication ID

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25 December 2017