Mechanisms underlying relaxation of rabbit aorta by BAY 41-2272, a nitric oxide-independent soluble guanylate cyclase activator. (Q46712818)

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7 January 2020

title

Mechanisms underlying relaxation of rabbit aorta by BAY 41-2272, a nitric oxide-independent soluble guanylate cyclase activator (English)

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Fernanda Bruschi Priviero

7 January 2020

author

1

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7 January 2020

Mário A Claudino

4

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7 January 2020

Gilberto de Nucci

5

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7 January 2020

Edson Antunes

6

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7 January 2020

author name string

Juliana S Baracat

2

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7 January 2020

Cleber E Teixeira

3

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publication date

1 September 2005

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Clinical and Experimental Pharmacology and Physiology

7 January 2020

published in

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7 January 2020

volume

32

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7 January 2020

issue

9

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7 January 2020

page(s)

728-734

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Nitric oxide and cyclic GMP formation upon electrical field stimulation cause relaxation of corpus cavernosum smooth muscle.

7 January 2020

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Purified soluble guanylyl cyclase expressed in a baculovirus/Sf9 system: stimulation by YC-1, nitric oxide, and carbon monoxide

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NO-independent regulatory site of direct sGC stimulators like YC-1 and BAY 41-2272

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Cardiorenal and humoral properties of a novel direct soluble guanylate cyclase stimulator BAY 41-2272 in experimental congestive heart failure

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Effects of the soluble guanylyl cyclase activator, YC-1, on vascular tone, cyclic GMP levels and phosphodiesterase activity

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NO- and haem-independent activation of soluble guanylyl cyclase: molecular basis and cardiovascular implications of a new pharmacological principle

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Release of nitric oxide from endothelial cells stimulated by YC-1, an activator of soluble guanylyl cyclase

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Relaxing effects induced by the soluble guanylyl cyclase stimulator BAY 41-2272 in human and rabbit corpus cavernosum

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Inhibitory effects on human eosinophil chemotaxis in vitro by BAY 41-2272, an activator of nitric oxide-independent site of soluble guanylate cyclase

21 January 2018

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Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscle.

21 January 2018

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Sodium nitroprusside-induced cGMP-independent vasodilator responses in the perfused rabbit ovarian vascular bed.

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Mechanism of cGMP contribution to the vasodilator response to NO in rat middle cerebral arteries

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Hypoxia enhances a cGMP-independent nitric oxide relaxing mechanism in pulmonary arteries

21 January 2018

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Soluble guanylyl cyclase activator YC-1 inhibits human neutrophil functions through a cGMP-independent but cAMP-dependent pathway

21 January 2018

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New insights into the intracellular mechanisms by which PGI2 analogues elicit vascular relaxation: cyclic AMP-independent, Gs-protein mediated-activation of MaxiK channel.

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Inhibition of extracellular Ca(2+) entry by YC-1, an activator of soluble guanylyl cyclase, through a cyclic GMP-independent pathway in rat neutrophils

21 January 2018

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Effect of PDE5 inhibition on coronary hemodynamics in pacing-induced heart failure

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The effect of sildenafil on nitric oxide-mediated vasodilation in healthy men.

21 January 2018

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10.1111/J.1440-1681.2005.04262.X

21 January 2018

Identifiers

DOI

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7 January 2020

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