The influence of dose and N-acetyltransferase-2 (NAT2) genotype and phenotype on the pharmacokinetics and pharmacodynamics of isoniazid. (Q46605137)

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30 December 2019

title

The influence of dose and N-acetyltransferase-2 (NAT2) genotype and phenotype on the pharmacokinetics and pharmacodynamics of isoniazid (English)

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author name string

P R Donald

1

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D P Parkin

2

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H I Seifart

3

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P D van Helden

5

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C J Werely

6

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F A Sirgel

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A Venter

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J S Maritz

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publication date

16 May 2007

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European Journal of Clinical Pharmacology

30 December 2019

published in

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volume

63

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page(s)

633-639

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issue

7

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https://scigraph.springernature.com/pub.10.1007/s00228-007-0305-5

30 December 2019

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Trimodality of isoniazid elimination: phenotype and genotype in patients with tuberculosis

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High-performance liquid chromatographic determination of isoniazid, acetylisoniazid and hydrazine in biological fluids

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Clinical implications of isoniazid blood levels in pulmonary tuberculosis

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Low isoniazid concentrations and outcome of tuberculosis treatment with once-weekly isoniazid and rifapentine

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The absorption, distribution, excretion, and short-term toxicity of isonicotinic acid hydrazide (nydrazid) in man

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Genotyping of the N-acetyltransferase2 polymorphism in the prediction of adverse drug reactions to isoniazid in Japanese patients

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Hydrazine derivatives of isonicotinic acid (rimifon marsilid) in the treatment of active progressive caseous-pneumonic tuberculosis; a preliminary report

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The prevention and treatment of isoniazid toxicity in the therapy of pulmonary tuberculosis: 1. An assessment of two vitamin B preparations and glutamic acid

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Should we use N-acetyltransferase type 2 genotyping to personalize isoniazid doses?

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Association between acquired rifamycin resistance and the pharmacokinetics of rifabutin and isoniazid among patients with HIV and tuberculosis

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12 December 2020

The influence of human N-acetyltransferase genotype on the early bactericidal activity of isoniazid

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Slow N-acetyltransferase 2 genotype affects the incidence of isoniazid and rifampicin-induced hepatotoxicity

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How drug resistance emerges as a result of poor compliance during short course chemotherapy for tuberculosis

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United States Public Health service cooperative investigation of antimicrobial therapy of tuberculosis, V. Report on thirty-two-week observations on combinations of isoniazid, streptomycin, and para-aminosalicylic acid

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The potential clinical significance of the isoniazid acetylator phenotype in the treatment of pulmonary tuberculosis

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On the metabolic fate of isoniazid

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10.1007/S00228-007-0305-5

Identifiers

DOI

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30 December 2019

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