Induction of centrosome and chromosome aberrations by imatinib in vitro (Q46577685)

23 December 2017

title

Induction of centrosome and chromosome aberrations by imatinib in vitro (English)

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23 December 2017

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5

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23 December 2017

author name string

Fabarius A

1

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23 December 2017

Giehl M

2

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23 December 2017

Frank O

3

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23 December 2017

Duesberg P

4

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23 December 2017

Hehlmann R

6

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23 December 2017

Seifarth W

7

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23 December 2017

publication date

1 September 2005

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23 December 2017

published in

Leukemia

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23 December 2017

volume

19

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23 December 2017

page(s)

1573-1578

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23 December 2017

issue

9

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23 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.leu.2403861

exact match

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cites work

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Philadelphia chromosome-negative cells with trisomy 8 after busulfan and interferon treatment of Ph1-positive chronic myelogenous leukemia

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Clonal Ph-negative hematopoiesis in CML after therapy with imatinib mesylate is frequently characterized by trisomy 8.

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Report of 34 patients with clonal chromosomal abnormalities in Philadelphia-negative cells during imatinib treatment of Philadelphia-positive chronic myeloid leukemia.

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Emergence of clonal cytogenetic abnormalities in Ph- cells in some CML patients in cytogenetic remission to imatinib but restoration of polyclonal hematopoiesis in the majority

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Clonal aberrations in Philadelphia chromosome negative hematopoiesis in patients with chronic myeloid leukemia treated with imatinib or interferon alpha

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Occurrence of additional chromosome aberrations in chronic myeloid leukemia patients treated with imatinib mesylate

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Demonstration of Philadelphia chromosome negative abnormal clones in patients with chronic myelogenous leukemia during major cytogenetic responses induced by imatinib mesylate.

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Centrosome aberrations in chronic myeloid leukemia correlate with stage of disease and chromosomal instability

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Centrosome aberrations--hen or egg in cancer initiation and progression?

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Imatinib mesylate (STI571) inhibits growth of primitive malignant progenitors in chronic myelogenous leukemia through reversal of abnormally increased proliferation

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p73 is regulated by tyrosine kinase c-Abl in the apoptotic response to DNA damage

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Functional interaction between DNA-PK and c-Abl in response to DNA damage

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Interaction between ATM protein and c-Abl in response to DNA damage

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Regulation of Rad51 function by c-Abl in response to DNA damage

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Instability of chromosome structure in cancer cells increases exponentially with degrees of aneuploidy

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Imatinib mesylate inhibits T-cell proliferation in vitro and delayed-type hypersensitivity in vivo

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Squamous cutaneous epithelial cell carcinoma in two CML patients with progressive disease under imatinib treatment

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The protein kinase complement of the human genome

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Overexpression of mammalian Rad51 does not stimulate tumorigenesis while a dominant-negative Rad51 affects centrosome fragmentation, ploidy and stimulates tumorigenesis, in p53-defective CHO cells.

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Genetic interactions between RAD51 and its paralogues for centrosome fragmentation and ploidy control, independently of the sensitivity to genotoxic stresses

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Centrosome amplification induced by DNA damage occurs during a prolonged G2 phase and involves ATM.

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Acute myeloblastic leukaemia without Philadelphia chromosome developing after interferon therapy for chronic myelocytic leukaemia with Philadelphia chromosome

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7 January 2021

Trisomy 8 in Philadelphia chromosome (Ph1)-negative cells in the course of Ph1-positive chronic myelocytic leukemia

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7 January 2021

A second case of trisomy 8 in Philadelphia chromosome (Ph)-negative cells during the course of Ph-positive chronic myelocytic leukemia

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7 January 2021

Determination of cell fate by c-Abl activation in the response to DNA damage

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7 January 2021

Identifiers

DOI

10.1038/SJ.LEU.2403861

1 reference

23 December 2017

PubMed ID

15990860

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23 December 2017