Postconditioning via stuttering reperfusion limits myocardial infarct size in rabbit hearts: role of ERK1/2. (Q46529109)

22 December 2017

title

Postconditioning via stuttering reperfusion limits myocardial infarct size in rabbit hearts: role of ERK1/2. (English)

1 reference

22 December 2017

1 reference

Chad E Darling

1

1 reference

22 December 2017

Rong Jiang

2

1 reference

22 December 2017

Michelle Maynard

3

1 reference

22 December 2017

Peter Whittaker

4

1 reference

22 December 2017

Jakob Vinten-Johansen

5

1 reference

22 December 2017

Karin Przyklenk

6

1 reference

22 December 2017

publication date

3 June 2005

1 reference

22 December 2017

American Journal of Physiology Heart and Circulatory Physiology

published in

1 reference

22 December 2017

volume

289

1 reference

22 December 2017

page(s)

H1618-26

1 reference

22 December 2017

issue

4

1 reference

22 December 2017

Acetylcholine, bradykinin, opioids, and phenylephrine, but not adenosine, trigger preconditioning by generating free radicals and opening mitochondrial K(ATP) channels

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Pharmacological manipulation of Ins(1,4,5)P3 signaling mimics preconditioning in rabbit heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

p38 MAPK activity is not increased early during sustained coronary artery occlusion in preconditioned versus control rabbit heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Myocardial protection with postconditioning is not enhanced by ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Postconditioning attenuates myocardial ischemia-reperfusion injury by inhibiting events in the early minutes of reperfusion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

ACh and adenosine activate PI3-kinase in rabbit hearts through transactivation of receptor tyrosine kinases

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

PI3 kinase and not p42/p44 appears to be implicated in the protection conferred by ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Primary and secondary signaling pathways in early preconditioning that converge on the mitochondria to produce cardioprotection

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Intracoronary L-arginine during reperfusion improves endothelial function and reduces infarct size

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Gradual reperfusion reduces infarct size and endothelial injury but augments neutrophil accumulation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Hypoxic postconditioning reduces cardiomyocyte loss by inhibiting ROS generation and intracellular Ca2+ overload

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Ischemic preconditioning activates phosphatidylinositol-3-kinase upstream of protein kinase C

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Postconditioning: a form of "modified reperfusion" protects the myocardium by activating the phosphatidylinositol 3-kinase-Akt pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Time course of endothelial dysfunction and myocardial injury during myocardial ischemia and reperfusion in the cat.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Postconditioning--A new link in nature's armor against myocardial ischemia-reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Pharmacological possibilities for protection against myocardial reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Postconditioning's protection is not dependent on circulating blood factors or cells but involves adenosine receptors and requires PI3-kinase and guanylyl cyclase activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Multiple, brief coronary occlusions during early reperfusion protect rabbit hearts by targeting cell signaling pathways.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Protecting the ischaemic and reperfused myocardium in acute myocardial infarction: distant dream or near reality?

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Rat and rabbit heart infarction: effects of anesthesia, perfusate, risk zone, and method of infarct sizing.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

Inhibition of myocardial injury by ischemic postconditioning during reperfusion: comparison with ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00055.2005

10.1152/AJPHEART.00055.2005

21 January 2018

Identifiers

DOI

1 reference

22 December 2017

PubMed ID

15937101

1 reference

22 December 2017