The pivotal role of high glucose-induced overexpression of PKCβ in the appearance of glucagon-like peptide-1 resistance in endothelial cells. (Q46470578)

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scientific article published on 19 November 2015

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English	The pivotal role of high glucose-induced overexpression of PKCβ in the appearance of glucagon-like peptide-1 resistance in endothelial cells.	scientific article published on 19 November 2015

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29 February 2020

The pivotal role of high glucose-induced overexpression of PKCβ in the appearance of glucagon-like peptide-1 resistance in endothelial cells (English)

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29 February 2020

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Antonio Ceriello

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29 February 2020

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29 February 2020

Valeria De Nigris

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29 February 2020

Stefano Genovese

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29 February 2020

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Lucia La Sala

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29 February 2020

Roberto Testa

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29 February 2020

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19 November 2015

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29 February 2020

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29 February 2020

54

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29 February 2020

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29 February 2020

396-410

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29 February 2020

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1 reference

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Effects of glucagon-like peptide-1 on endothelial function in type 2 diabetes patients with stable coronary artery disease.

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The possible protective role of glucagon-like peptide 1 on endothelium during the meal and evidence for an "endothelial resistance" to glucagon-like peptide 1 in diabetes

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Examining the effects of hyperglycemia on pancreatic endocrine function in humans: evidence for in vivo glucotoxicity.

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Glucagon like peptide-1-induced glucose metabolism in differentiated human muscle satellite cells is attenuated by hyperglycemia

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Glucagon-like peptides: regulators of cell proliferation, differentiation, and apoptosis

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Cardiovascular benefits of GLP-1-based herapies in patients with diabetes mellitus type 2: effects on endothelial and vascular dysfunction beyond glycemic control

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Cardiovascular consequences of drugs used for the treatment of diabetes: potential promise of incretin-based therapies

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FoxOs at the crossroads of cellular metabolism, differentiation, and transformation

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Evolutionary paths of the cAMP-dependent protein kinase (PKA) catalytic subunits

1 reference

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21 January 2018

Chronic hyperglycemia downregulates GLP-1 receptor signaling in pancreatic β-cells via protein kinase A

1 reference

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21 January 2018

Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway

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The antioxidant defense system Keap1-Nrf2 comprises a multiple sensing mechanism for responding to a wide range of chemical compounds

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Molecular mechanisms activating the Nrf2-Keap1 pathway of antioxidant gene regulation

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Nrf2-Keap1 regulation of cellular defense mechanisms against electrophiles and reactive oxygen species

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Antioxidant effects of diallyl trisulfide on high glucose-induced apoptosis are mediated by the PI3K/Akt-dependent activation of Nrf2 in cardiomyocytes

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21 January 2018

Endoplasmic reticulum stress in beta-cells and development of diabetes

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21 January 2018

ER stress in adipocytes inhibits insulin signaling, represses lipolysis, and alters the secretion of adipokines without inhibiting glucose transport.

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21 January 2018

Endoplasmic reticulum stress-induced apoptosis in the development of diabetes: is there a role for adipose tissue and liver?

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21 January 2018

Lipopolysaccharide, high glucose and saturated fatty acids induce endoplasmic reticulum stress in cultured primary human adipocytes: Salicylate alleviates this stress

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21 January 2018

Diabetes, hyperglycemia and accelerated atherosclerosis: evidence supporting a role for endoplasmic reticulum (ER) stress signaling

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21 January 2018

Endothelial dysfunction and diabetes: roles of hyperglycemia, impaired insulin signaling and obesity

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21 January 2018

Exendin-4 attenuates endoplasmic reticulum stress through a SIRT1-dependent mechanism

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Exendin-4 attenuates high glucose-induced cardiomyocyte apoptosis via inhibition of endoplasmic reticulum stress and activation of SERCA2a

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21 January 2018

The human glucagon-like peptide-1 analogue liraglutide preserves pancreatic beta cells via regulation of cell kinetics and suppression of oxidative and endoplasmic reticulum stress in a mouse model of diabetes

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21 January 2018

Endothelial dysfunction in diabetes mellitus: possible involvement of endoplasmic reticulum stress?

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Glucose exposure pattern determines glucagon-like peptide 1 receptor expression and signaling through endoplasmic reticulum stress in rat insulinoma cells

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1 reference

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21 January 2018

GLP-1 analogue, Liraglutide protects human umbilical vein endothelial cells against high glucose induced endoplasmic reticulum stress

1 reference

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GLP-1 ameliorates the proliferation activity of INS-1 cells inhibited by intermittent high glucose concentrations through the regulation of cyclins

1 reference

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GLP-1 receptor activation improves beta cell function and survival following induction of endoplasmic reticulum stress

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Glucagon-Like Peptide-1 Analog Liraglutide Protects against Diabetic Cardiomyopathy by the Inhibition of the Endoplasmic Reticulum Stress Pathway

1 reference

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Downregulation of GLP-1 and GIP receptor expression by hyperglycemia: possible contribution to impaired incretin effects in diabetes

1 reference

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Glucagon-like peptide-1 (GLP-1) inhibits advanced glycation end product (AGE)-induced up-regulation of VCAM-1 mRNA levels in endothelial cells by suppressing AGE receptor (RAGE) expression

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1 reference

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Identifiers

10.1007/S12020-015-0799-Z

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26585565%20AND%20SRC:MED&resulttype=core&format=json

29 February 2020

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:26585565%20AND%20SRC:MED&resulttype=core&format=json

29 February 2020

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