Rac1 mediates sex difference in cardiac tumor necrosis factor-alpha expression via NADPH oxidase-ERK1/2/p38 MAPK pathway in endotoxemia. (Q46007197)

18 December 2017

title

Rac1 mediates sex difference in cardiac tumor necrosis factor-alpha expression via NADPH oxidase-ERK1/2/p38 MAPK pathway in endotoxemia. (English)

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18 December 2017

author name string

Huaqing Zhu

series ordinal

1

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18 December 2017

Limei Shan

series ordinal

2

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18 December 2017

Tianqing Peng

series ordinal

3

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18 December 2017

publication date

18 May 2009

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18 December 2017

Journal of Molecular and Cellular Cardiology

published in

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18 December 2017

volume

47

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18 December 2017

issue

2

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18 December 2017

page(s)

264-274

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18 December 2017

Endotoxin-Induced Myocardial Tumor Necrosis Factor-α Synthesis Depresses Contractility of Isolated Rat Hearts

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Pathophysiologically relevant concentrations of tumor necrosis factor-alpha promote progressive left ventricular dysfunction and remodeling in rats

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Inhibition of p38 MAPK decreases myocardial TNF-alpha expression and improves myocardial function and survival in endotoxemia

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Pivotal role of gp91phox-containing NADH oxidase in lipopolysaccharide-induced tumor necrosis factor-alpha expression and myocardial depression

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The neutrophil NADPH oxidase

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Lipopolysaccharide induces Rac1-dependent reactive oxygen species formation and coordinates tumor necrosis factor-alpha secretion through IKK regulation of NF-kappa B.

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Down-regulation of Rac-1 GTPase by Estrogen

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7 January 2021

Identifiers

DOI

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18 December 2017

PubMed ID

19450605

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18 December 2017