Oxidative stress triggers neuronal caspase-independent death: endonuclease G involvement in programmed cell death-type III. (Q45937554)

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19582370%20AND%20SRC:MED&resulttype=core&format=json

2 February 2020

title

Oxidative stress triggers neuronal caspase-independent death: endonuclease G involvement in programmed cell death-type III (English)

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2 February 2020

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1

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2 February 2020

author name string

Philip M Beart

series ordinal

2

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19582370%20AND%20SRC:MED&resulttype=core&format=json

2 February 2020

Phillip Nagley

series ordinal

3

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2 February 2020

publication date

7 July 2009

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Cellular and Molecular Life Sciences

2 February 2020

published in

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19582370%20AND%20SRC:MED&resulttype=core&format=json

2 February 2020

volume

66

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2 February 2020

issue

16

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2 February 2020

page(s)

2773-2787

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https://scigraph.springernature.com/pub.10.1007/s00018-009-0079-2

2 February 2020

exact match

0 references

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Oxidative stress and Parkinson's disease

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Oxidative stress and energy crises in neuronal dysfunction

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Calpain mediates excitotoxic DNA fragmentation via mitochondrial pathways in adult brains: evidence from calpastatin mutant mice.

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Mammalian mitochondrial endonuclease G. Digestion of R-loops and localization in intermembrane space

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Hierarchical recruitment by AMPA but not staurosporine of pro-apoptotic mitochondrial signaling in cultured cortical neurons: evidence for caspase-dependent/independent cross-talk

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BNIP3 upregulation and EndoG translocation in delayed neuronal death in stroke and in hypoxia

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Differential insult-dependent recruitment of the intrinsic mitochondrial pathway during neuronal programmed cell death

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Failure to complete apoptosis following neonatal hypoxia-ischemia manifests as "continuum" phenotype of cell death and occurs with multiple manifestations of mitochondrial dysfunction in rodent forebrain

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Necrostatin-1 protects against glutamate-induced glutathione depletion and caspase-independent cell death in HT-22 cells

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Caspase-7 expanded function and intrinsic expression level underlies strain-specific brain phenotype of caspase-3-null mice.

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Mitochondrial membrane potential and glutamate excitotoxicity in cultured cerebellar granule cells.

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Primers for mitochondrial DNA replication generated by endonuclease G.

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Apoptosis-inducing factor substitutes for caspase executioners in NMDA-triggered excitotoxic neuronal death.

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Effect of peroxide, sodium, and calcium on brain mitochondrial respiration in vitro: potential role in cerebral ischemia and reperfusion

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Regulation of the apoptosis-necrosis switch

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Oxidative stress and neuronal death/survival signaling in cerebral ischemia

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Switch from caspase-dependent to caspase-independent death during heart development: essential role of endonuclease G in ischemia-induced DNA processing of differentiated cardiomyocytes

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Cytochrome c maintains mitochondrial transmembrane potential and ATP generation after outer mitochondrial membrane permeabilization during the apoptotic process

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Hypoxia ischemia-mediated cell death in neonatal rat brain

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Oxidative stress induces caspase-independent retinal apoptosis in vitro

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Apoptosis-inducing factor is involved in the regulation of caspase-independent neuronal cell death

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12 December 2020

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The harlequin mouse mutation downregulates apoptosis-inducing factor

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Molecular characterization of mitochondrial apoptosis-inducing factor

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12 December 2020

Endonuclease G: a mitochondrial protein released in apoptosis and involved in caspase-independent DNA degradation.

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12 December 2020

10.1007/S00018-009-0079-2

Identifiers

DOI

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:19582370%20AND%20SRC:MED&resulttype=core&format=json

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2 February 2020

1007627072

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release_yrw7p5lcjfdsrlwrb4lotoohrq

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24 November 2022

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