Coenzyme Q10 and remacemide hydrochloride ameliorate motor deficits in a Huntington's disease transgenic mouse model (Q45304699)

11 December 2017

title

Coenzyme Q10 and remacemide hydrochloride ameliorate motor deficits in a Huntington's disease transgenic mouse model (English)

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11 December 2017

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Schilling G

1

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11 December 2017

Coonfield ML

series ordinal

2

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11 December 2017

Ross CA

series ordinal

3

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11 December 2017

Borchelt DR

series ordinal

4

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11 December 2017

language of work or name

English

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publication date

1 November 2001

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11 December 2017

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11 December 2017

volume

315

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11 December 2017

issue

3

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11 December 2017

page(s)

149-153

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Mitochondrial dysfunction in neurodegenerative diseases

11 December 2017

cites work

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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

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A randomized, placebo-controlled trial of coenzyme Q10 and remacemide in Huntington's disease

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Coenzyme Q10 administration increases brain mitochondrial concentrations and exerts neuroprotective effects

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Inhibition of caspase-1 slows disease progression in a mouse model of Huntington's disease

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Huntington's Disease and Dentatorubral‐Pallidoluysian Atrophy: Proteins, Pathogenesis and Pathology

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Polyglutamine-expanded huntingtin promotes sensitization of N-methyl-D-aspartate receptors via post-synaptic density 95.

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Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo

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Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin

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Mitochondrial dysfunction and free radical damage in the Huntington R6/2 transgenic mouse

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10.1016/S0304-3940(01)02326-6

Identifiers

DOI

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11 December 2017

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