18F-FDG PET uptake in the pre-Huntington disease caudate affects the time-to-onset independently of CAG expansion size (Q45288484)

11 December 2017

title

18F-FDG PET uptake in the pre-Huntington disease caudate affects the time-to-onset independently of CAG expansion size (English)

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Fluorodeoxyglucose Positron Emission Tomography

11 December 2017

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6

Ferdinando Squitieri

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11 December 2017

Andrea Ciarmiello

1

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11 December 2017

Giampiero Giovacchini

2

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11 December 2017

Sara Orobello

3

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11 December 2017

Laura Bruselli

4

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11 December 2017

Francesca Elifani

5

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11 December 2017

publication date

12 April 2012

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11 December 2017

number of pages

7

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European Journal of Nuclear Medicine and Molecular Imaging

inferred from page(s)

published in

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11 December 2017

volume

39

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11 December 2017

issue

6

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11 December 2017

page(s)

1030-1036

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Serial changes of cerebral glucose metabolism and caudate size in persons at risk for Huntington's disease.

11 December 2017

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Striatal glucose metabolism and dopamine D2 receptor binding in asymptomatic gene carriers and patients with Huntington's disease

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Informed consent: study of quality of information given to participants in a clinical trial

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Homozygosity for CAG mutation in Huntington disease is associated with a more severe clinical course

21 January 2018

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21 January 2018

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21 January 2018

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Reversal of neuropathology and motor dysfunction in a conditional model of Huntington's disease

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Onset and rate of striatal atrophy in preclinical Huntington disease

21 January 2018

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Longitudinal analysis of regional grey matter loss in Huntington disease: effects of the length of the expanded CAG repeat.

21 January 2018

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Distinct brain volume changes correlating with clinical stage, disease progression rate, mutation size, and age at onset prediction as early biomarkers of brain atrophy in Huntington's disease

21 January 2018

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Selective defect of in vivo glycolysis in early Huntington's disease striatum

21 January 2018

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Expression of mutant huntingtin in glial cells contributes to neuronal excitotoxicity

21 January 2018

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Huntingtin controls neurotrophic support and survival of neurons by enhancing BDNF vesicular transport along microtubules

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Early defect of transforming growth factor β1 formation in Huntington's disease.

21 January 2018

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DNA instability in postmitotic neurons

21 January 2018

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DNA instability in replicating Huntington's disease lymphoblasts

21 January 2018

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Somatic expansion of the Huntington's disease CAG repeat in the brain is associated with an earlier age of disease onset

21 January 2018

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A genome scan for modifiers of age at onset in Huntington disease: The HD MAPS study.

21 January 2018

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12 December 2020

Huntingtin aggregate-associated axonal degeneration is an early pathological event in Huntington's disease mice.

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12 December 2020

The likelihood of being affected with Huntington disease by a particular age, for a specific CAG size

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12 December 2020

Brain white-matter volume loss and glucose hypometabolism precede the clinical symptoms of Huntington's disease.

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12 December 2020

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12 December 2020

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12 December 2020

Phenotypic characterization of individuals with 30-40 CAG repeats in the Huntington disease (HD) gene reveals HD cases with 36 repeats and apparently normal elderly individuals with 36-39 repeats

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12 December 2020

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Identifiers

DOI

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Dimensions Publication ID

11 December 2017

1034271569

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