P53 plays a protective role against UV- and cisplatin-induced apoptosis in transcription-coupled repair proficient fibroblasts. (Q44953358)

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3 December 2019

title

P53 plays a protective role against UV- and cisplatin-induced apoptosis in transcription-coupled repair proficient fibroblasts (English)

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3 December 2019

1

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3 December 2019

author name string

Becerril C

series ordinal

2

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3 December 2019

Ljungman M

series ordinal

3

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3 December 2019

language of work or name

English

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publication date

1 October 2001

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3 December 2019

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3 December 2019

volume

20

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3 December 2019

page(s)

6805-6808

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3 December 2019

issue

46

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1204901

3 December 2019

exact match

0 references

cites work

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Proneness to UV-induced apoptosis in human fibroblasts defective in transcription coupled repair is associated with the lack of Mdm2 transactivation

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Expression of wild-type p53 is required for efficient global genomic nucleotide excision repair in UV-irradiated human fibroblasts

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Transformed cells require continuous activity of RNA polymerase II to resist oncogene-induced apoptosis

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Effect of ultraviolet light, methyl methanesulfonate and ionizing radiation on the genotoxic response and apoptosis of mouse fibroblasts lacking c-Fos, p53 or both

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Primary mouse fibroblasts deficient for c-Fos, p53 or for both proteins are hypersensitive to UV light and alkylating agent-induced chromosomal breakage and apoptosis

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p53, the cellular gatekeeper for growth and division

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UV light-induced degradation of RNA polymerase II is dependent on the Cockayne's syndrome A and B proteins but not p53 or MLH1.

21 January 2018

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The tumor suppressor p53 can both stimulate and inhibit ultraviolet light-induced apoptosis

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Wildtype p53 is required for heat shock and ultraviolet light enhanced repair of a UV-damaged reporter gene

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Role for p53 in the recovery of transcription and protection against apoptosis induced by ultraviolet light

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Persistent DNA damage induced by ultraviolet light inhibits p21waf1 and bax expression: implications for DNA repair, UV sensitivity and the induction of apoptosis

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Capacity of UV-irradiated human fibroblasts to support adenovirus DNA synthesis correlates with transcription-coupled repair and is reduced in SV40-transformed cells and cells expressing mutant p53.

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RNA polymerase II stalled at a thymine dimer: footprint and effect on excision repair

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p53-mediated protective responses to UV irradiation.

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Human cells compromised for p53 function exhibit defective global and transcription-coupled nucleotide excision repair, whereas cells compromised for pRb function are defective only in global repair

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The role of the E6-p53 interaction in the molecular pathogenesis of HPV.

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Deficient repair of the transcribed strand of active genes in Cockayne's syndrome cells

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p53 modulation of TFIIH-associated nucleotide excision repair activity

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Influence of p53 tumor suppressor protein on bias of DNA repair and apoptotic response in human cells

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Inhibition of RNA Polymerase II Transcription in Human Cell Extracts by Cisplatin DNA Damage

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Transcription abnormalities potentiate apoptosis of normal human fibroblasts.

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7 January 2021

p53-degradation by HPV-16 E6 preferentially affects the removal of cyclobutane pyrimidine dimers from non-transcribed strand and sensitizes mammary epithelial cells to UV-irradiation

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7 January 2021

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7 January 2021

p53 and the Li-Fraumeni syndrome

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7 January 2021

DNA adducts of cis-diamminedichloroplatinum(II) and its trans isomer inhibit RNA polymerase II differentially in vivo

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Faulty DNA polymerase delta/epsilon-mediated excision repair in response to gamma radiation or ultraviolet light in p53-deficient fibroblast strains from affected members of a cancer-prone family with Li-Fraumeni syndrome

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7 January 2021