Role of the AP-1 element and redox factor-1 (Ref-1) in mediating transcriptional induction of DT-diaphorase gene expression by oltipraz: a target for chemoprevention (Q44485480)

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English	Role of the AP-1 element and redox factor-1 (Ref-1) in mediating transcriptional induction of DT-diaphorase gene expression by oltipraz: a target for chemoprevention	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

3 December 2017

Role of the AP-1 element and redox factor-1 (Ref-1) in mediating transcriptional induction of DT-diaphorase gene expression by oltipraz: a target for chemoprevention (English)

1 reference

Europe PubMed Central

PubMed publication ID

3 December 2017

author name string

Kang-Shen Yao

1

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3 December 2017

Peter J O'Dwyer

2

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Europe PubMed Central

PubMed publication ID

3 December 2017

language of work or name

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publication date

1 July 2003

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Europe PubMed Central

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3 December 2017

Biochemical Pharmacology

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3 December 2017

66

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Europe PubMed Central

PubMed publication ID

3 December 2017

1

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Europe PubMed Central

PubMed publication ID

3 December 2017

15-23

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Europe PubMed Central

PubMed publication ID

3 December 2017

Inhibitory effects of 5-(2-pyrazinyl)-4-methyl-1,2-dithiol-3-thione (Oltipraz) on carcinogenesis induced by benzo[a]pyrene, diethylnitrosamine and uracil mustard.

1 reference

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Chemopreventive activity of Oltipraz against N-nitrosobis(2-oxopropyl)amine (BOP)-induced ductal pancreatic carcinoma development and effects on survival of Syrian golden hamsters

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Effect of oltipraz [5-(2-pyrazinyl)-4-methyl-1,2-dithiol-3-thione] on azoxymethane-induced biochemical changes related to early colon carcinogenesis in male F344 rats.

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Biochemical effects of dithiolthiones

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1,2-Dithiol-3-thione analogs: effects on NAD(P)H:quinone reductase and glutathione levels in murine hepatoma cells.

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Increase of NAD(P)H:quinone reductase by dietary antioxidants: possible role in protection against carcinogenesis and toxicity

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Identification of a common chemical signal regulating the induction of enzymes that protect against chemical carcinogenesis

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Characterization of xenobiotic responsive elements upstream from the drug-metabolizing cytochrome P-450c gene: a similarity to glucocorticoid regulatory elements

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Regulation of glutathione S-transferase Ya subunit gene expression: identification of a unique xenobiotic-responsive element controlling inducible expression by planar aromatic compounds

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Xenobiotic-inducible expression of murine glutathione S-transferase Ya subunit gene is controlled by an electrophile-responsive element

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Glutathione S-transferases: gene structure and regulation of expression

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Antitumor promotion by phenolic antioxidants: inhibition of AP-1 activity through induction of Fra expression

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Activation of AP-1 and of a nuclear redox factor, Ref-1, in the response of HT29 colon cancer cells to hypoxia

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Involvement of NF-kappa B in the induction of NAD(P)H:quinone oxidoreductase (DT-diaphorase) by hypoxia, oltipraz and mitomycin C.

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Oncogene jun encodes a sequence-specific trans-activator similar to AP-1.

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Structure of the FBJ Murine Osteosarcoma Virus Genome: Molecular Cloning of Its Associated Helper Virus and the Cellular Homolog of the v-fos Gene from Mouse and Human Cells

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A gene activated by growth factors is related to the oncogene v-jun

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Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei

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fra-1: a serum-inducible, cellular immediate-early gene that encodes a fos-related antigen.

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Isolation of cDNA clones encoding an enzyme from bovine cells that repairs oxidative DNA damagein vitro: homology with bacterial repair enzymes

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A glutathione transferase in human leukocytes as a marker for the susceptibility to lung cancer

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Human NAD(P)H:quinone oxidoreductase (NQO1) gene structure and induction by dioxin

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Nrf1 and Nrf2 positively and c-Fos and Fra1 negatively regulate the human antioxidant response element-mediated expression of NAD(P)H:quinone oxidoreductase1 gene

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Sensitivity to carcinogenesis is increased and chemoprotective efficacy of enzyme inducers is lost in nrf2 transcription factor-deficient mice

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An 'environment to nucleus' signaling system operates in B lymphocytes: redox status modulates BSAP/Pax-5 activation through Ref-1 nuclear translocation

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Ref-1 controls pax-8 DNA-binding activity

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Activation of hypoxia-inducible transcription factor depends primarily upon redox-sensitive stabilization of its alpha subunit

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Ref-1 regulates the transactivation and pro-apoptotic functions of p53 in vivo.

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Two different cellular redox systems regulate the DNA-binding activity of the p50 subunit of NF-kappa B in vitro

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Overexpression of Ref-1 inhibits hypoxia and tumor necrosis factor-induced endothelial cell apoptosis through nuclear factor-kappab-independent and -dependent pathways

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The drug efflux pump MRP2: regulation of expression in physiopathological situations and by endogenous and exogenous compounds

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Modulation of gene expression in subjects at risk for colorectal cancer by the chemopreventive dithiolethione oltipraz

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Regulation of phase 2 enzyme induction by oltipraz and other dithiolethiones

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Relation of structure of curcumin analogs to their potencies as inducers of Phase 2 detoxification enzymes

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10.1016/S0006-2952(03)00163-1

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Europe PubMed Central

PubMed publication ID

3 December 2017

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3 December 2017

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