2-Methoxyestradiol induces apoptosis in Ewing sarcoma cells through mitochondrial hydrogen peroxide production (Q44429627)

2 December 2017

2-Methoxyestradiol induces apoptosis in Ewing sarcoma cells through mitochondrial hydrogen peroxide production (English)

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2 December 2017

Mojgan Djavaheri-Mergny

object named as

Mojgan Djavaheri-Mergny

series ordinal

1

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author name string

Juana Wietzerbin

series ordinal

2

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2 December 2017

Françoise Besançon

series ordinal

3

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2 December 2017

language of work or name

English

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publication date

1 May 2003

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2 December 2017

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2 December 2017

22

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2 December 2017

17

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2 December 2017

2558-2567

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1206356

2 December 2017

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cites work

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2-Methoxyestradiol-induced phosphorylation of Bcl-2: uncoupling from JNK/SAPK activation.

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Mitochondrial redox signaling during apoptosis

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Quantitation of mitochondrial alterations associated with apoptosis

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Regulation and measurement of oxidative stress in apoptosis

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Novel role for JNK as a stress-activated Bcl2 kinase

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DIABLO promotes apoptosis by removing MIHA/XIAP from processed caspase 9

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alpha-Synuclein protects against oxidative stress via inactivation of the c-Jun N-terminal kinase stress-signaling pathway in neuronal cells

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2-methoxyestradiol blocks cell-cycle progression at G(2)/M phase and inhibits growth of human prostate cancer cells

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How effective is dose-intensive/myeloablative therapy against Ewing's sarcoma/primitive neuroectodermal tumor metastatic to bone or bone marrow? The Memorial Sloan-Kettering experience and a literature review

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Ebselen, a glutathione peroxidase mimetic seleno-organic compound, as a multifunctional antioxidant. Implication for inflammation-associated carcinogenesis

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Involvement of mitochondrial phospholipid hydroperoxide glutathione peroxidase as an antiapoptotic factor

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2-Methoxyestradiol induces G2/M arrest and apoptosis in prostate cancer

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The physiological estrogen metabolite 2-methoxyestradiol reduces tumor growth and induces apoptosis in human solid tumors

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Redox control of mitochondrial functions

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Ebselen: a substrate for human thioredoxin reductase strongly stimulating its hydroperoxide reductase activity and a superfast thioredoxin oxidant

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Overexpression of MnSOD protects murine fibrosarcoma cells (FSa-II) from apoptosis and promotes a differentiation program upon treatment with 5-azacytidine: involvement of MAPK and NFkappaB pathways

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Role of redox potential and reactive oxygen species in stress signaling

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Direct activation of mitochondrial apoptosis machinery by c-Jun N-terminal kinase in adult cardiac myocytes

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Death receptors: signaling and modulation

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Gene fusion with an ETS DNA-binding domain caused by chromosome translocation in human tumours

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The endogenous oestrogen metabolite 2-methoxyoestradiol inhibits angiogenesis and suppresses tumour growth

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Free radicals, lipid peroxidation, and cell damage

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Interactions of 2-methoxyestradiol, an endogenous mammalian metabolite, with unpolymerized tubulin and with tubulin polymers

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Superoxide dismutase as a target for the selective killing of cancer cells

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NF-kappa B activation results in rapid inactivation of JNK in TNF alpha-treated Ewing sarcoma cells: a mechanism for the anti-apoptotic effect of NF-kappa B

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Doxorubicin-induced apoptosis in endothelial cells and cardiomyocytes is ameliorated by nitrone spin traps and ebselen. Role of reactive oxygen and nitrogen species

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Induction of apoptosis in human lung cancer cells after wild-type p53 activation by methoxyestradiol

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2-Methoxyestradiol induces mitochondria dependent apoptotic signaling in pancreatic cancer cells

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Role of reactive oxygen species (ROS) in apoptosis induction

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Activation of caspase-9 is required for UV-induced apoptosis of human keratinocytes

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Identifiers

DOI

10.1038/SJ.ONC.1206356

1 reference

Dimensions Publication ID

2 December 2017

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1 reference