Kainic acid induces 14-3-3 zeta expression in distinct regions of rat brain (Q44212272)

30 November 2017

Kainic acid induces 14-3-3 zeta expression in distinct regions of rat brain (English)

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30 November 2017

Marcel Patrick van der Brug

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1

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30 November 2017

Sharon Goodenough

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2

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30 November 2017

Peter Wilce

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3

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30 November 2017

language of work or name

English

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publication date

1 November 2002

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30 November 2017

6

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inferred from page(s)

published in

Brain Research

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30 November 2017

956

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30 November 2017

1

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30 November 2017

110-115

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14-3-3 proteins: biological function and domain structure

30 November 2017

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A paradox after systemic kainate injection in rats: lesser damage of hippocampal CA1 neurons after higher doses

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Leonardo, a Drosophila 14-3-3 protein involved in learning, regulates presynaptic function.

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Integration of calcium and cyclic AMP signaling pathways by 14-3-3

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14-3-3 zeta negatively regulates raf-1 activity by interactions with the Raf-1 cysteine-rich domain

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Is c-Jun involved in nerve cell death following status epilepticus and hypoxic-ischaemic brain injury?

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Regulation of p53 downstream genes.

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Increased expression of mitochondrial genes in human alcoholic brain revealed by differential display.

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14-3-3 protein homologs required for the DNA damage checkpoint in fission yeast.

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Cell death and immunohistochemistry of p53, c-Fos and c-Jun after spermine injection into the rat striatum

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Immediate early gene expression and delayed cell death in limbic areas of the rat brain after kainic acid treatment and recovery in the cold.

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14-3-3sigma is a p53-regulated inhibitor of G2/M progression

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Kainate-evoked secondary gene expression in the rat hippocampus

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Status epilepticus induces p53 sequence-specific DNA binding in mature rat brain.

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14-3-3 proteins: regulation of subcellular localization by molecular interference

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Enhanced expression of 14-3-3 family members in injured motoneurons.

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Crystal structure of the 14-3-3zeta:serotonin N-acetyltransferase complex. a role for scaffolding in enzyme regulation

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Differential expression of apolipoprotein D and apolipoprotein E in the kainic acid-lesioned rat hippocampus.

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Cell death during development of the nervous system

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Novel differentially expressed genes induced by kainic acid in hippocampus: putative molecular effectors of plasticity and injury.

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14-3-3zeta binds a phosphorylated Raf peptide and an unphosphorylated peptide via its conserved amphipathic groove

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Cell death, gliosis, and synaptic remodeling in the hippocampus of epileptic rats.

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Induction of tumor suppressor p53 and DNA fragmentation in organotypic hippocampal cultures following excitotoxin treatment.

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14-3-3 proteins associate with A20 in an isoform-specific manner and function both as chaperone and adapter molecules

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Kainic acid-induced gene expression in the rat hippocampus is severely affected by adrenalectomy.

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Mutations in the hydrophobic surface of an amphipathic groove of 14-3-3zeta disrupt its interaction with Raf-1 kinase.

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ATM-dependent activation of p53 involves dephosphorylation and association with 14-3-3 proteins

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14-3-3 proteins block apoptosis and differentially regulate MAPK cascades

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The proapoptotic protein Bad binds the amphipathic groove of 14-3-3zeta

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Residues of 14-3-3 zeta required for activation of exoenzyme S of Pseudomonas aeruginosa.

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AP-1 and Egr DNA-binding activities are increased in rat brain during ethanol withdrawal.

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7 January 2021

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Identifiers

DOI

10.1016/S0006-8993(02)03487-X

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30 November 2017

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