Focal glomerulosclerosis in proviral and c-fms transgenic mice links Vpr expression to HIV-associated nephropathy (Q43577959)

24 November 2017

title

Focal glomerulosclerosis in proviral and c-fms transgenic mice links Vpr expression to HIV-associated nephropathy (English)

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24 November 2017

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6

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24 November 2017

Peter Dickie

1

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24 November 2017

Amanda Roberts

2

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24 November 2017

Richard Uwiera

3

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24 November 2017

Jennifer Witmer

4

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24 November 2017

Kirti Sharma

5

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24 November 2017

language of work or name

English

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publication date

1 April 2004

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24 November 2017

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24 November 2017

volume

322

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24 November 2017

issue

1

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24 November 2017

page(s)

69-81

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The HIV-1 Vpr co-activator induces a conformational change in TFIIB.

24 November 2017

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Distinct effects in primary macrophages and lymphocytes of the human immunodeficiency virus type 1 accessory genes vpr, vpu, and nef: mutational analysis of a primary HIV-1 isolate

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HIV-1 kills renal tubular epithelial cells in vitro by triggering an apoptotic pathway involving caspase activation and Fas upregulation

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Vpr is required for efficient replication of human immunodeficiency virus type-1 in mononuclear phagocytes

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HIV type 1 Nef perturbs eye lens development in transgenic mice

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Chemokine receptor (CCR5) expression in human kidneys and in the HIV infected macaque

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HIV infects glomerular endothelial and mesangial but not epithelial cells in vitro

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Glomerulosclerosis and viral gene expression in HIV-transgenic mice: Role of nef

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Viral DNA in microdissected renal biopsy tissue from HIV infected patients with nephrotic syndrome

7 January 2021

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The HIV-1 virion-associated protein vpr is a coactivator of the human glucocorticoid receptor

7 January 2021

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HIV-1 expression induces cyclin D1 expression and pRb phosphorylation in infected podocytes: cell-cycle mechanisms contributing to the proliferative phenotype in HIV-associated nephropathy

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7 January 2021

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Human immunodeficiency virus type 1 (HIV-1) Vpr functions as an immediate-early protein during HIV-1 infection

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7 January 2021

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7 January 2021

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Identifiers

DOI

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24 November 2017

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