Targeted GLUT-4 deficiency in the heart induces cardiomyocyte hypertrophy and impaired contractility linked with Ca(2+) and proton flux dysregulation (Q43229407)

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scientific article published on 3 December 2009

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English	Targeted GLUT-4 deficiency in the heart induces cardiomyocyte hypertrophy and impaired contractility linked with Ca(2+) and proton flux dysregulation	scientific article published on 3 December 2009

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scholarly article

1 reference

Europe PubMed Central

16 November 2017

Targeted GLUT-4 deficiency in the heart induces cardiomyocyte hypertrophy and impaired contractility linked with Ca(2+) and proton flux dysregulation (English)

1 reference

Europe PubMed Central

16 November 2017

Lea M Delbridge

6

object named as

Lea M D Delbridge

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Europe PubMed Central

16 November 2017

author name string

Andrea A Domenighetti

1

1 reference

Europe PubMed Central

16 November 2017

Vennetia R Danes

2

1 reference

Europe PubMed Central

16 November 2017

Claire L Curl

3

1 reference

Europe PubMed Central

16 November 2017

Jennifer M Favaloro

4

1 reference

Europe PubMed Central

16 November 2017

Joseph Proietto

5

1 reference

Europe PubMed Central

16 November 2017

publication date

3 December 2009

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Europe PubMed Central

16 November 2017

Journal of Molecular and Cellular Cardiology

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Europe PubMed Central

16 November 2017

48

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Europe PubMed Central

16 November 2017

4

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Europe PubMed Central

16 November 2017

663-672

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Europe PubMed Central

16 November 2017

Diabetic cardiomyopathy: mechanisms, diagnosis and treatment.

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Diabetic cardiomyopathy: the search for a unifying hypothesis

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Diabetic cardiomyopathy revisited

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Insulin-resistant cardiomyopathy clinical evidence, mechanisms, and treatment options

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Myocardial insulin resistance and cardiac complications of diabetes.

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Early abnormalities of cardiac function in non-insulin-dependent diabetes mellitus and impaired glucose tolerance

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Diabetic cardiomyopathy

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Decreased sarcoplasmic reticulum activity and contractility in diabetic db/db mouse heart

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Metabolic disturbances in diabetic cardiomyopathy

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Growth hormone inhibition of glucose uptake in adipocytes occurs without affecting GLUT4 translocation through an insulin receptor substrate-2-phosphatidylinositol 3-kinase-dependent pathway

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Diabetic cardiomyopathy: evidence, mechanisms, and therapeutic implications

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Myocardial glucose transport and utilization in patients with type 2 diabetes mellitus, left ventricular dysfunction, and coronary artery disease.

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Downregulation of myocardial myocyte enhancer factor 2C and myocyte enhancer factor 2C-regulated gene expression in diabetic patients with nonischemic heart failure

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Insulin resistance in patients with cardiac hypertrophy.

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Initial steps of insulin signaling and glucose transport are defective in the type 2 diabetic rat heart

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Insulin resistance in adult cardiomyocytes undergoing dedifferentiation: role of GLUT4 expression and translocation

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Stimulus interval-dependent differences in Ca2+ transients and contractile responses of diabetic rat cardiomyocytes

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Threshold effects of glucose transporter-4 (GLUT4) deficiency on cardiac glucose uptake and development of hypertrophy.

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Functional and metabolic remodelling in GLUT4-deficient hearts confers hyper-responsiveness to substrate intervention

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Angiotensin II-mediated phenotypic cardiomyocyte remodeling leads to age-dependent cardiac dysfunction and failure.

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Steady-state twitch Ca2+ fluxes and cytosolic Ca2+ buffering in rabbit ventricular myocytes

1 reference

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Upregulation of Na(+)/Ca(2+) exchanger expression and function in an arrhythmogenic rabbit model of heart failure

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Relationship between intracellular pH and tension development in resting ventricular muscle and myocytes

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Characterization of intracellular pH regulation in the guinea-pig ventricular myocyte.

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Chronic inhibition of Na+/H+-exchanger attenuates cardiac hypertrophy and prevents cellular remodeling in heart failure

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Enhanced activity of ventricular Na+-HCO3- cotransport in pressure overload hypertrophy

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Modulation by pH0 and intracellular Ca2+ of Na(+)-H+ exchange in diabetic rat isolated ventricular myocytes

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The pathogenesis of myocardial fibrosis in the setting of diabetic cardiomyopathy

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The antioxidant tempol inhibits cardiac hypertrophy in the insulin-resistant GLUT4-deficient mouse in vivo

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Direct effects of high glucose and insulin on protein synthesis in cultured cardiac myocytes and DNA and collagen synthesis in cardiac fibroblasts

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Functional coupling between glycolysis and sarcoplasmic reticulum Ca2+ transport

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Effects of Na(+)/Ca(2+)-exchanger overexpression on excitation-contraction coupling in adult rabbit ventricular myocytes.

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Overexpression of the Na(+)/Ca(2+) exchanger and inhibition of the sarcoplasmic reticulum Ca(2+)-ATPase in ventricular myocytes from transgenic mice

1 reference

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7 January 2021

Cardiac hypertrophy with preserved contractile function after selective deletion of GLUT4 from the heart

1 reference

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Responses of GLUT4-deficient hearts to ischemia underscore the importance of glycolysis

1 reference

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Recipes for creating animal models of diabetic cardiovascular disease

1 reference

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Impaired cardiac contractile function in ventricular myocytes from leptin-deficient ob/ob obese mice

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Cardiac contractile dysfunction in Lep/Lep obesity is accompanied by NADPH oxidase activation, oxidative modification of sarco(endo)plasmic reticulum Ca2+-ATPase and myosin heavy chain isozyme switch

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Fidarestat improves cardiomyocyte contractile function in db/db diabetic obese mice through a histone deacetylase Sir2-dependent mechanism

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Insulin and inositol 1,4,5-trisphosphate trigger abnormal cytosolic Ca2+ transients and reveal mitochondrial Ca2+ handling defects in cardiomyocytes of ob/ob mice.

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Mechanisms of [Ca2+]i transient decrease in cardiomyopathy of db/db type 2 diabetic mice

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Impact of Type 2 diabetes and aging on cardiomyocyte function and O-linked N-acetylglucosamine levels in the heart

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Cardiomyocyte dysfunction in insulin-resistant rats: a female advantage.

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Impaired SERCA function contributes to cardiomyocyte dysfunction in insulin resistant rats

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Enhanced activity of the myocardial Na+/H+ exchanger contributes to left ventricular hypertrophy in the Goto-Kakizaki rat model of type 2 diabetes: critical role of Akt

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Myofilament sensitivity to Ca2+ in ventricular myocytes from the Goto-Kakizaki diabetic rat.

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pHi regulation in myocardium of the spontaneously hypertensive rat. Compensated enhanced activity of the Na(+)-H+ exchanger

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Sodium-hydrogen exchanger, cardiac overload, and myocardial hypertrophy

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pH-Dependence of extrinsic and intrinsic H(+)-ion mobility in the rat ventricular myocyte, investigated using flash photolysis of a caged-H(+) compound.

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Identification of sodium-calcium exchange current in single ventricular cells of guinea-pig

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Temperature dependence of Na+-H+ exchange, Na+-HCO3- co-transport, intracellular buffering and intracellular pH in guinea-pig ventricular myocytes.

1 reference

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Altered calcium homeostasis does not explain the contractile deficit of diabetic cardiomyopathy.

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H(2)O(2)-induced Ca(2+) overload in NRVM involves ERK1/2 MAP kinases: role for an NHE-1-dependent pathway

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7 January 2021

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10.1016/J.YJMCC.2009.11.017

1 reference

Europe PubMed Central

16 November 2017

1 reference

Europe PubMed Central

16 November 2017

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