Mitochondrial effects with ceramide-induced cardiac apoptosis are different from those of palmitate (Q42596013)

3 November 2017

Mitochondrial effects with ceramide-induced cardiac apoptosis are different from those of palmitate (English)

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3 November 2017

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Jennifer Y Kong

1

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PubMed ID

12667483

3 November 2017

Simon W Rabkin

series ordinal

2

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PubMed ID

12667483

3 November 2017

language of work or name

English

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publication date

1 April 2003

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Archives of Biochemistry and Biophysics

3 November 2017

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3 November 2017

412

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3 November 2017

196-206

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3 November 2017

2

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Signal transduction of stress via ceramide

3 November 2017

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Doxorubicin induces slow ceramide accumulation and late apoptosis in cultured adult rat ventricular myocytes

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Glucose and palmitic acid induce degeneration of myofibrils and modulate apoptosis in rat adult cardiomyocytes

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The voltage-independent B-type Ca2+ channel modulates apoptosis of cardiac myocytes

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Involvement of caspase 3- and 8-like proteases in ceramide-induced apoptosis of cardiomyocytes

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Production and metabolism of ceramide in normal and ischemic-reperfused myocardium of rats

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Ceramide induces hepatocyte cell death through disruption of mitochondrial function in the rat.

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Direct Effect of Ceramide on the Mitochondrial Electron Transport Chain Leads to Generation of Reactive Oxygen Species

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Ceramide Induces CytochromecRelease from Isolated Mitochondria

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Functional consequences of the sustained or transient activation by Bax of the mitochondrial permeability transition pore

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The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis

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Implication of mitochondrial hydrogen peroxide generation in ceramide-induced apoptosis

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The cytotoxicity of tumor necrosis factor depends on induction of the mitochondrial permeability transition

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Ordering of ceramide formation, caspase activation, and mitochondrial changes during CD95- and DNA damage-induced apoptosis

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Inhibition of carnitine palmitoyltransferase I augments sphingolipid synthesis and palmitate-induced apoptosis

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An update of the enzymology and regulation of sphingomyelin metabolism

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Mitochondria and apoptosis

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The mitochondrion in apoptosis: how Pandora's box opens

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Mitochondrial respiratory chain inhibitors induce apoptosis.

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Recent progress on regulation of the mitochondrial permeability transition pore; a cyclosporin-sensitive pore in the inner mitochondrial membrane

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Sequential reduction of mitochondrial transmembrane potential and generation of reactive oxygen species in early programmed cell death

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Breaking the mitochondrial barrier

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The mitochondrial apoptosome: a killer unleashed by the cytochrome seas

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Intracellular heterogeneity in mitochondrial membrane potentials revealed by a J-aggregate-forming lipophilic cation JC-1.

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Cytochrome c-mediated apoptosis in cells lacking mitochondrial DNA. Signaling pathway involving release and caspase 3 activation is conserved

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Evaluation of fluorescent dyes for the detection of mitochondrial membrane potential changes in cultured cardiomyocytes

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Features of apoptotic cells measured by flow cytometry

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Capsaicin-induced depolarisation of mitochondria in dorsal root ganglion neurons is enhanced by vanilloid receptors

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Palmitate-induced apoptosis can occur through a ceramide-independent pathway

7 January 2021

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Cytofluorometric detection of mitochondrial alterations in early CD95/Fas/APO-1-triggered apoptosis of Jurkat T lymphoma cells. Comparison of seven mitochondrion-specific fluorochromes.

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Analysis of mitochondrial morphology and function with novel fixable fluorescent stains

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The use of chloromethyl-X-rosamine (Mitotracker red) to measure loss of mitochondrial membrane potential in apoptotic cells is incompatible with cell fixation

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Detection of apoptosis and changes in mitochondrial membrane potential with chloromethyl-X-rosamine

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Synthetic ceramides induce growth arrest or apoptosis by altering cellular redox status

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Ceramide Interaction with the Respiratory Chain of Heart Mitochondria†

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Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

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Mitochondrial permeability transition and swelling can occur reversibly without inducing cell death in intact human cells

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Palmitic acid opens a novel cyclosporin A-insensitive pore in the inner mitochondrial membrane

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Fatty acid oxidation in embryonic chick tissues

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Interaction of fatty acid and glucose oxidation by cultured heart cells

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Inhibition of Ca2(+)-induced large-amplitude swelling of liver and heart mitochondria by cyclosporin is probably caused by the inhibitor binding to mitochondrial-matrix peptidyl-prolyl cis-trans isomerase and preventing it interacting with the adeni

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Inhibition of mitochondrial respiratory chain complex I by TNF results in cytochrome c release, membrane permeability transition, and apoptosis.

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Modulation of palmitate-induced cardiomyocyte cell death by interventions that alter intracellular calcium

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Lack of mitochondrial anionic phospholipids causes an inhibition of translation of protein components of the electron transport chain. A yeast genetic model system for the study of anionic phospholipid function in mitochondria

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10.1016/S0003-9861(03)00008-0

7 January 2021

Identifiers

DOI

1 reference

3 November 2017

1 reference

PubMed ID

12667483