Subacute systemic 3-nitropropionic acid intoxication induces a distinct motor disorder in adult C57Bl/6 mice: behavioural and histopathological characterisation (Q42526865)

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English	Subacute systemic 3-nitropropionic acid intoxication induces a distinct motor disorder in adult C57Bl/6 mice: behavioural and histopathological characterisation	scientific article

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scholarly article

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Europe PubMed Central

1 November 2017

Subacute systemic 3-nitropropionic acid intoxication induces a distinct motor disorder in adult C57Bl/6 mice: behavioural and histopathological characterisation (English)

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Europe PubMed Central

1 November 2017

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Tison F

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Europe PubMed Central

1 November 2017

author name string

Fernagut PO

1

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Europe PubMed Central

1 November 2017

Diguet E

2

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Europe PubMed Central

1 November 2017

Stefanova N

3

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Europe PubMed Central

1 November 2017

Biran M

4

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Europe PubMed Central

1 November 2017

Wenning GK

5

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Europe PubMed Central

1 November 2017

Canioni P

6

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Europe PubMed Central

1 November 2017

Bioulac B

7

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Europe PubMed Central

1 November 2017

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publication date

1 January 2002

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Europe PubMed Central

1 November 2017

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Europe PubMed Central

1 November 2017

114

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Europe PubMed Central

1 November 2017

4

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Europe PubMed Central

1 November 2017

1005-1017

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Europe PubMed Central

1 November 2017

Metabolic compromise with systemic 3-nitropropionic acid produces striatal apoptosis in Sprague-Dawley rats but not in BALB/c ByJ mice

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Malonate and 3-nitropropionic acid neurotoxicity are reduced in transgenic mice expressing a caspase-1 dominant-negative mutant.

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Mice with a partial deficiency of manganese superoxide dismutase show increased vulnerability to the mitochondrial toxins malonate, 3-nitropropionic acid, and MPTP.

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Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid

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Limited-time exposure to mitochondrial toxins may lead to chronic progressive neurodegenerative diseases

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Cronbach's alpha

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Increased vulnerability to 3-nitropropionic acid in an animal model of Huntington's disease.

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Behavioral pathology induced by repeated systemic injections of 3-nitropropionic acid mimics the motoric symptoms of Huntington's disease

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Systemic, but not intraparenchymal, administration of 3-nitropropionic acid mimics the neuropathology of Huntington's disease: a speculative explanation

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Effects of chronic MPTP and 3-nitropropionic acid in nonhuman primates

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Partial inhibition of brain succinate dehydrogenase by 3-nitropropionic acid is sufficient to initiate striatal degeneration in rat.

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Temporal evolution of 3-nitropropionic acid-induced neurodegeneration in the rat brain by T2-weighted, diffusion-weighted, and perfusion magnetic resonance imaging.

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Effects of MPTP on locomotor activity in mice

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Basal ganglia degeneration, myelin alterations, and enzyme inhibition induced in mice by the plant toxin 3-nitropropanoic acid

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Exploration of the dopamine transporter: in vitro and in vivo characterization of a high-affinity and high-specificity iodinated tropane derivative (E)-N-(3-iodoprop-2-enyl)-2beta-carbomethoxy-3beta-(4'-m ethylph enyl)nortropane (PE2I).

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Quantifiable bradykinesia, gait abnormalities and Huntington's disease-like striatal lesions in rats chronically treated with 3-nitropropionic acid

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Mice transgenic for the Huntington's disease mutation are resistant to chronic 3-nitropropionic acid-induced striatal toxicity.

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10.1016/S0306-4522(02)00205-1

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Europe PubMed Central

1 November 2017

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Europe PubMed Central

1 November 2017

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