Changes in localization of human discs large (hDlg) during keratinocyte differentiation are [corrected] associated with expression of alternatively spliced hDlg variants. (Q42514187)

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English	Changes in localization of human discs large (hDlg) during keratinocyte differentiation are [corrected] associated with expression of alternatively spliced hDlg variants.	scientific article

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Changes in localization of human discs large (hDlg) during keratinocyte differentiation are [corrected] associated with expression of alternatively spliced hDlg variants (English)

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Vincenzo Calautti

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Vanderweil S

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Nguyen HO

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Foley A

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Baden HP

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Viel A

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24 May 2007

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Experimental Cell Research

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2521-2530

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Signaling pathways are focused at specialized regions of the plasma membrane by scaffolding proteins of the MAGUK family

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Dlg protein is required for junction structure, cell polarity, and proliferation control in Drosophila epithelia

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Craniofacial dysmorphogenesis including cleft palate in mice with an insertional mutation in the discs large gene

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Zonula adherens formation in Caenorhabditis elegans requires dlg-1, the homologue of the Drosophila gene discs large

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DLG-1 is a MAGUK similar to SAP97 and is required for adherens junction formation

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Requirement of PDZ-containing proteins for cell cycle regulation and differentiation in the mouse lens epithelium

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Cdc42 and Par6-PKCzeta regulate the spatially localized association of Dlg1 and APC to control cell polarization.

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Mutagenesis of the epithelial polarity gene, discs large 1, perturbs nephrogenesis in the developing mouse kidney

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Inhibition of patterned cell shape change and cell invasion by Discs large during Drosophila oogenesis

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The APC-hDLG complex negatively regulates cell cycle progression from the G0/G1 to S phase

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Molecular cloning of the lethal(1)discs large-1 oncogene of Drosophila.

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Functional expression of rat synapse-associated proteins SAP97 and SAP102 in Drosophila dlg-1 mutants: effects on tumor suppression and synaptic bouton structure.

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HPV E6 specifically targets different cellular pools of its PDZ domain-containing tumour suppressor substrates for proteasome-mediated degradation

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Differential expression of the human homologue of drosophila discs large oncosuppressor in histologic samples from human papillomavirus-associated lesions as a marker for progression to malignancy

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Changes in expression of the human homologue of the Drosophila discs large tumour suppressor protein in high-grade premalignant cervical neoplasias

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Somatic mutations and altered expression of the candidate tumor suppressors CSNK1 epsilon, DLG1, and EDD/hHYD in mammary ductal carcinoma

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Binding of high-risk human papillomavirus E6 oncoproteins to the human homologue of the Drosophila discs large tumor suppressor protein

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Binding of human virus oncoproteins to hDlg/SAP97, a mammalian homolog of the Drosophila discs large tumor suppressor protein

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Oncogenic human papillomavirus E6 proteins target the discs large tumour suppressor for proteasome-mediated degradation

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Cloning and characterization of hdlg: the human homologue of the Drosophila discs large tumor suppressor binds to protein 4.1

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Human homolog of disc-large is required for adherens junction assembly and differentiation of human intestinal epithelial cells

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Colocalization of APC and DLG at the tips of cellular protrusions in cultured epithelial cells and its dependency on cytoskeletons.

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Redistribution of the discs large tumor suppressor protein during mitosis

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The distribution and function of alternatively spliced insertions in hDlg

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Identification of brain-specific splicing variants of the hDLG1 gene and altered splicing in neuroblastoma cell lines

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Two independent domains of hDlg are sufficient for subcellular targeting: the PDZ1-2 conformational unit and an alternatively spliced domain

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Oncogenic function for the Dlg1 mammalian homolog of the Drosophila discs-large tumor suppressor

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Human discs large and scrib are localized at the same regions in colon mucosa and changes in their expression patterns are correlated with loss of tissue architecture during malignant progression.

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Differential expression of human Dlg in cervical intraepithelial neoplasias

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The expression of desmoglein isoforms in cultured human keratinocytes is regulated by calcium, serum, and protein kinase C

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Modular organization of the PDZ domains in the human discs-large protein suggests a mechanism for coupling PDZ domain-binding proteins to ATP and the membrane cytoskeleton

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