Foreign metallothionein-I expression by transient transfection in MT-I and MT-II null astrocytes confers increased protection against acute methylmercury cytotoxicity (Q42482831)

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scientific article published on February 2000

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English	Foreign metallothionein-I expression by transient transfection in MT-I and MT-II null astrocytes confers increased protection against acute methylmercury cytotoxicity	scientific article published on February 2000

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scholarly article

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Europe PubMed Central

PubMed publication ID

1 November 2017

Foreign metallothionein-I expression by transient transfection in MT-I and MT-II null astrocytes confers increased protection against acute methylmercury cytotoxicity (English)

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Europe PubMed Central

PubMed publication ID

1 November 2017

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author name string

C P Yao

1

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1 November 2017

J W Allen

2

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1 November 2017

L A Mutkus

3

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1 November 2017

S B Xu

4

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1 November 2017

K H Tan

5

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1 November 2017

M Aschner

6

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1 November 2017

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publication date

1 February 2000

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1 November 2017

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1 November 2017

855

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1 November 2017

1

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Europe PubMed Central

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1 November 2017

32-38

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Europe PubMed Central

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1 November 2017

Selective modulation of NMDA responses by reduction and oxidation

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Induction of astrocyte metallothioneins (MTs) by zinc confers resistance against the acute cytotoxic effects of methylmercury on cell swelling, Na+ uptake, and K+ release.

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Astrocyte and neuron coculturing method.

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Metallothioneins in brain--the role in physiology and pathology.

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Uptake of methylmercury in the rat brain: effects of amino acids

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Methylmercury uptake in rat primary astrocyte cultures: the role of the neutral amino acid transport system.

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Intracellular glutathione (GSH) levels modulate mercuric chloride (MC)- and methylmercuric chloride (MeHgCl)-induced amino acid release from neonatal rat primary astrocytes cultures

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A hypothesis regarding the protective role of metallothioneins against the toxicity of DNA interactive anticancer drugs

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Differential expression of the metallothionein gene in liver and brain of mice and rats.

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Metallothionein induction by bismuth in neonatal rat primary astrocyte cultures.

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Analysis of the effects of overexpression of metallothionein-I in transgenic mice on the reproductive toxicology of cadmium

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Transcriptional induction of the mouse metallothionein-I gene in hydrogen peroxide-treated Hepa cells involves a composite major late transcription factor/antioxidant response element and metal response promoter elements

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Metallothionein

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Metallothionein and glutathione as determinants of cellular retention and extrusion of cadmium and mercury

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Modification of methylmercury toxicity and metabolism by selenium and vitamin E: possible mechanisms

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Metallothionein

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A physicochemical rationale for the biological activity of mercury and its compounds

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Characterization of metallothionein-I-transgenic mice

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Induction of metallothionein mRNA and protein in murine astrocyte cultures

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Transgenic mouse blastocysts that overexpress metallothionein-I resist cadmium toxicity in vitro

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Effects of butyrate homologues on metallothionein induction in rat primary hepatocyte cultures

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Transgenic mice that overexpress metallothionein-I are protected from cadmium lethality and hepatotoxicity.

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Targeted disruption of metallothionein I and II genes increases sensitivity to cadmium

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Targeting and germ-line transmission of a null mutation at the metallothionein I and II loci in mouse

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Oxidative damage to nucleic acids in motor neurons containing mercury

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Metallothionein induction in neonatal rat primary astrocyte cultures protects against methylmercury cytotoxicity

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Yeast and mammalian metallothioneins functionally substitute for yeast copper-zinc superoxide dismutase

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3 Toxicological significance of metallothionein

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Identifiers

10.1016/S0006-8993(99)02211-8

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 November 2017

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