Mycobacterium tuberculosis PstS1 amplifies IFN-γ and induces IL-17/IL-22 responses by unrelated memory CD4+ T cells via dendritic cell activation (Q42261100)

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scientific article published on 15 July 2013

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English	Mycobacterium tuberculosis PstS1 amplifies IFN-γ and induces IL-17/IL-22 responses by unrelated memory CD4+ T cells via dendritic cell activation	scientific article published on 15 July 2013

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

19 October 2017

Mycobacterium tuberculosis PstS1 amplifies IFN-γ and induces IL-17/IL-22 responses by unrelated memory CD4+ T cells via dendritic cell activation (English)

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Europe PubMed Central

PubMed publication ID

19 October 2017

Mycobacterium tuberculosis

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Fabrizio Mattei

object named as

Fabrizio Mattei

4

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Giovanna Schiavoni

object named as

Giovanna Schiavoni

2

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object named as

Carla Palma

1

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object named as

Lucia Gabriele

9

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author name string

Laura Abalsamo

3

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Europe PubMed Central

PubMed publication ID

19 October 2017

Giovanni Piccaro

5

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Europe PubMed Central

PubMed publication ID

19 October 2017

Massimo Sanchez

6

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Europe PubMed Central

PubMed publication ID

19 October 2017

Carmen Fernandez

7

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Europe PubMed Central

PubMed publication ID

19 October 2017

Mahavir Singh

8

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Europe PubMed Central

PubMed publication ID

19 October 2017

language of work or name

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publication date

15 July 2013

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Europe PubMed Central

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19 October 2017

European Journal of Immunology

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Europe PubMed Central

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19 October 2017

43

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Europe PubMed Central

PubMed publication ID

19 October 2017

9

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Europe PubMed Central

PubMed publication ID

19 October 2017

2386-2397

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Europe PubMed Central

PubMed publication ID

19 October 2017

Disseminated tuberculosis in interferon gamma gene-disrupted mice

1 reference

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Properties and protective value of the secondary versus primary T helper type 1 response to airborne Mycobacterium tuberculosis infection in mice

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Mycobacterium bovisBCG-Specific Th17 Cells Confer Partial Protection againstMycobacterium tuberculosisInfection in the Absence of Gamma Interferon

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Pathological role of interleukin 17 in mice subjected to repeated BCG vaccination after infection with Mycobacterium tuberculosis

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Alphabeta T cell receptor-positive cells and interferon-gamma, but not inducible nitric oxide synthase, are critical for granuloma necrosis in a mouse model of mycobacteria-induced pulmonary immunopathology

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Interferon-gamma-dependent mechanisms of mycobacteria-induced pulmonary immunopathology: the role of angiostasis and CXCR3-targeted chemokines for granuloma necrosis.

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Bystander activation of CD8+ T lymphocytes during experimental mycobacterial infection.

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Bystander activation of CD8+ T cells contributes to the rapid production of IFN-gamma in response to bacterial pathogens

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The PE and PPE proteins of Mycobacterium tuberculosis.

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PE_PGRS antigens of Mycobacterium tuberculosis induce maturation and activation of human dendritic cells.

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Mycobacterium tuberculosis with disruption in genes encoding the phosphate binding proteins PstS1 and PstS2 is deficient in phosphate uptake and demonstrates reduced in vivo virulence

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Effect of growth state on transcription levels of genes encoding major secreted antigens of Mycobacterium tuberculosis in the mouse lung

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The M. tuberculosis phosphate-binding lipoproteins PstS1 and PstS3 induce Th1 and Th17 responses that are not associated with protection against M. tuberculosis infection

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Immunodominant PstS1 antigen of mycobacterium tuberculosis is a potent biological response modifier for the treatment of bladder cancer

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Mucosal administration of Ag85B-ESAT-6 protects against infection with Mycobacterium tuberculosis and boosts prior bacillus Calmette-Guerin immunity.

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Regulation of TH17 cell differentiation by innate immune signals

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Differential antigen processing by dendritic cell subsets in vivo

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Syk- and CARD9-dependent coupling of innate immunity to the induction of T helper cells that produce interleukin 17

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Potent and selective stimulation of memory-phenotype CD8+ T cells in vivo by IL-15.

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Human CD4+ memory T cells are preferential targets for bystander activation and apoptosis.

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IL-15 mimics T cell receptor crosslinking in the induction of cellular proliferation, gene expression, and cytotoxicity in CD8+ memory T cells

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Heterologous T cell immunity in severe hepatitis C virus infection

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Clinical value of the measurement of Mycobacterium tuberculosis specific antibody in pulmonary tuberculosis

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Essential role of IL-17A in the formation of a mycobacterial infection-induced granuloma in the lung

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Role of 4-1BB receptor in the control played by CD8(+) T cells on IFN-gamma production by Mycobacterium tuberculosis antigen-specific CD4(+) T Cells

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21 January 2018

Distinct, specific IL-17- and IL-22-producing CD4+ T cell subsets contribute to the human anti-mycobacterial immune response

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21 January 2018

Predominance of interleukin-22 over interleukin-17 at the site of disease in human tuberculosis

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21 January 2018

Attrition of T cell memory: selective loss of LCMV epitope-specific memory CD8 T cells following infections with heterologous viruses

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21 January 2018

Pathogen-induced human TH17 cells produce IFN-γ or IL-10 and are regulated by IL-1β.

1 reference

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21 January 2018

Interleukin (IL)-15 and IL-7 jointly regulate homeostatic proliferation of memory phenotype CD8+ cells but are not required for memory phenotype CD4+ cells

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Interferon-gamma-inducing factor enhances T helper 1 cytokine production by stimulated human T cells: synergism with interleukin-12 for interferon-gamma production

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10.1002/EJI.201243245

1 reference

Europe PubMed Central

PubMed publication ID

19 October 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

19 October 2017

ResearchGate publication ID

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