IFNL3 mediates interaction between innate immune cells: Implications for hepatitis C virus pathogenesis. (Q42247379)

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22 February 2020

title

IFNL3 mediates interaction between innate immune cells: Implications for hepatitis C virus pathogenesis (English)

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22 February 2020

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22 February 2020

Golo Ahlenstiel

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22 February 2020

Vijay Suppiah

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22 February 2020

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Jacob George

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22 February 2020

author name string

Kate S O'Connor

1

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22 February 2020

Stephen Schibeci

4

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22 February 2020

Adrian Ong

5

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22 February 2020

David van der Poorten

7

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22 February 2020

Mark W Douglas

8

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22 February 2020

Martin D Weltman

9

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22 February 2020

Graeme J Stewart

10

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22 February 2020

Christopher Liddle

11

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22 February 2020

David R Booth

13

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22 February 2020

publication date

17 September 2013

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22 February 2020

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22 February 2020

volume

20

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22 February 2020

issue

6

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22 February 2020

page(s)

598-605

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Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance

22 February 2020

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IL-28, IL-29 and their class II cytokine receptor IL-28R

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Despite IFN-lambda receptor expression, blood immune cells, but not keratinocytes or melanocytes, have an impaired response to type III interferons: implications for therapeutic applications of these cytokines

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Lambda interferon inhibits hepatitis B and C virus replication

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21 January 2018

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21 January 2018

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A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus

21 January 2018

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