Propofol potentiates ATP-activated currents of recombinant P2X(4) receptor channels expressed in human embryonic kidney 293 cells (Q40885274)

24 September 2017

title

Propofol potentiates ATP-activated currents of recombinant P2X(4) receptor channels expressed in human embryonic kidney 293 cells (English)

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24 September 2017

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A Tomioka

1

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24 September 2017

S Ueno

2

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24 September 2017

K Kohama

3

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24 September 2017

F Goto

4

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24 September 2017

K Inoue

5

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24 September 2017

language of work or name

English

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publication date

1 April 2000

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24 September 2017

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24 September 2017

volume

284

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24 September 2017

issue

3

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24 September 2017

page(s)

167-170

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Propofol modulates the effects of chemoconvulsants acting at GABAergic, glycinergic, and glutamate receptor subtypes

24 September 2017

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Unilateral convulsion after induction of anaesthesia with propofol.

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Cloning OF P2X5 and P2X6 receptors and the distribution and properties of an extended family of ATP-gated ion channels

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Distinct ATP receptors on pain-sensing and stretch-sensing neurons.

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The actions of propofol on inhibitory amino acid receptors of bovine adrenomedullary chromaffin cells and rodent central neurones

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7 January 2021

Coexpression of P2X2 and P2X3 receptor subunits can account for ATP-gated currents in sensory neurons

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7 January 2021

P2X4: an ATP-activated ionotropic receptor cloned from rat brain

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7 January 2021

In vivo pathway of thermal hyperalgesia by intrathecal administration of alpha,beta-methylene ATP in mouse spinal cord: involvement of the glutamate-NMDA receptor system

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Bicuculline and gabazine are allosteric inhibitors of channel opening of the GABAA receptor

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Cell type-specific ATP-activated responses in rat dorsal root ganglion neurons

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A new class of ligand-gated ion channel defined by P2X receptor for extracellular ATP

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Effects of propofol on various AMPA-, kainate- and NMDA-selective glutamate receptor channels expressed in Xenopus oocytes

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The effect of thiopental and propofol on NMDA- and AMPA-mediated glutamate excitotoxicity.

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Identifiers

DOI

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24 September 2017

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