The BH3 domain is required for caspase-independent cell death induced by Bax and oligomycin (Q40885189)

24 September 2017

The BH3 domain is required for caspase-independent cell death induced by Bax and oligomycin (English)

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24 September 2017

Fitch ME

1

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24 September 2017

Chang CM

series ordinal

2

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24 September 2017

Parslow TG

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3

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24 September 2017

1 April 2000

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Cell Death & Differentiation

24 September 2017

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24 September 2017

7

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24 September 2017

4

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24 September 2017

338-349

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4400659

24 September 2017

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cites work

Mitochondria and apoptosis

1 reference

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

21 January 2018

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

21 January 2018

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Bcl-2 inhibits the mitochondrial release of an apoptogenic protease

21 January 2018

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Molecular characterization of mitochondrial apoptosis-inducing factor

21 January 2018

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Mitochondrial release of caspase-2 and -9 during the apoptotic process

21 January 2018

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Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarization.

21 January 2018

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The Bcl-2 protein family: arbiters of cell survival

21 January 2018

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BCL-2 family: regulators of cell death

21 January 2018

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The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

21 January 2018

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Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

21 January 2018

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Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

21 January 2018

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Movement of Bax from the cytosol to mitochondria during apoptosis

21 January 2018

1 reference

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Bax directly induces release of cytochrome c from isolated mitochondria

21 January 2018

1 reference

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Bax interacts with the permeability transition pore to induce permeability transition and cytochrome c release in isolated mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

BAX-induced cell death may not require interleukin 1 beta-converting enzyme-like proteases

21 January 2018

1 reference

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Structure-function comparisons of the proapoptotic protein Bax in yeast and mammalian cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Interaction of E1B 19K with Bax is required to block Bax-induced loss of mitochondrial membrane potential and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Solution structure of BID, an intracellular amplifier of apoptotic signaling

21 January 2018

1 reference

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A peptide sequence from Bax that converts Bcl-2 into an activator of apoptosis

21 January 2018

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Heterodimerization-independent functions of cell death regulatory proteins Bax and Bcl-2 in yeast and mammalian cells

21 January 2018

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Differential requirement for caspase 9 in apoptotic pathways in vivo.

21 January 2018

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Reduced apoptosis and cytochrome c-mediated caspase activation in mice lacking caspase 9

21 January 2018

1 reference

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Inhibition of Bax channel-forming activity by Bcl-2

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Comparison of the ion channel characteristics of proapoptotic BAX and antiapoptotic BCL-2.

21 January 2018

1 reference

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Targeted disruption of mouse conventional kinesin heavy chain, kif5B, results in abnormal perinuclear clustering of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

The Mitochondrial F0F1-ATPase proton pump is required for function of the proapoptotic protein Bax in yeast and mammalian cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Bax and adenine nucleotide translocator cooperate in the mitochondrial control of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

The overexpression of Bax produces cell death upon induction of the mitochondrial permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4400659

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7 January 2021

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Identifiers

DOI

10.1038/SJ.CDD.4400659

1 reference

Dimensions Publication ID

24 September 2017

0 references

1 reference