Overexpression of human alpha-synuclein causes dopamine neuron death in rat primary culture and immortalized mesencephalon-derived cells. (Q40877706)

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Overexpression of human α-synuclein causes dopamine neuron death in rat primary culture and immortalized mesencephalon-derived cells

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English	Overexpression of human alpha-synuclein causes dopamine neuron death in rat primary culture and immortalized mesencephalon-derived cells.	scientific article	Overexpression of human α-synuclein causes dopamine neuron death in rat primary culture and immortalized mesencephalon-derived cells

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7 November 2019

Overexpression of human alpha-synuclein causes dopamine neuron death in rat primary culture and immortalized mesencephalon-derived cells (English)

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7 November 2019

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7 November 2019

author name string

W Zhou

1

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J Schaack

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K N Prasad

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C R Freed

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1 June 2000

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7 November 2019

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33-43

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7 November 2019

Characterization and transplantation of two neuronal cell lines with dopaminergic properties

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Genetic analysis of families with Parkinson disease that carry the Ala53Thr mutation in the gene encoding alpha-synuclein

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Immortalized dopamine neurons: A model to study neurotoxicity and neuroprotection

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Improvement of neurological deficits in 6-hydroxydopamine-lesioned rats after transplantation with allogeneic simian virus 40 large tumor antigen gene-induced immortalized dopamine cells

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The synucleins: a family of proteins involved in synaptic function, plasticity, neurodegeneration and disease.

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Accelerated in vitro fibril formation by a mutant alpha-synuclein linked to early-onset Parkinson disease.

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Aggregates from mutant and wild-type alpha-synuclein proteins and NAC peptide induce apoptotic cell death in human neuroblastoma cells by formation of beta-sheet and amyloid-like filaments

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Effects of the mutations Ala30 to Pro and Ala53 to Thr on the physical and morphological properties of alpha-synuclein protein implicated in Parkinson's disease

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Synphilin-1 associates with alpha-synuclein and promotes the formation of cytosolic inclusions

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Adenovirus-mediated expression of an olfactory cyclic nucleotide-gated channel regulates the endogenous Ca2+-inhibitable adenylyl cyclase in C6-2B glioma cells.

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Characterization of a novel protein regulated during the critical period for song learning in the zebra finch.

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Mutant and wild type human alpha-synucleins assemble into elongated filaments with distinct morphologies in vitro

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Neuropathology in movement disorders

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The relevance of the Lewy body to the pathogenesis of idiopathic Parkinson's disease

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7 January 2021

Mechanism of 6-hydroxydopamine neurotoxicity.

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Characteristics of a human cell line transformed by DNA from human adenovirus type 5

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Methods for construction of adenovirus vectors

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Human recombinant NACP/alpha-synuclein is aggregated and fibrillated in vitro: relevance for Lewy body disease

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Oxidative stress induces amyloid-like aggregate formation of NACP/alpha-synuclein in vitro

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The precursor protein of non-A beta component of Alzheimer's disease amyloid is a presynaptic protein of the central nervous system

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Nigral and cortical Lewy bodies and dystrophic nigral neurites in Parkinson's disease and cortical Lewy body disease contain alpha-synuclein immunoreactivity.

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Binding of Abeta to alpha- and beta-synucleins: identification of segments in alpha-synuclein/NAC precursor that bind Abeta and NAC

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Ala30Pro mutation in the gene encoding alpha-synuclein in Parkinson's disease

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Alpha synuclein in neurodegenerative disorders: murderer or accomplice?

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Toxicity of 6-hydroxydopamine and dopamine for dopaminergic neurons in culture.

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Both familial Parkinson's disease mutations accelerate alpha-synuclein aggregation

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Self-oligomerization of NACP, the precursor protein of the non-amyloid beta/A4 protein (A beta) component of Alzheimer's disease amyloid, observed in the presence of a C-terminal A beta fragment (residues 25-35).

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Developmental expression of alpha-synuclein in rat hippocampus and cerebral cortex.

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Mutation in the alpha-synuclein gene identified in families with Parkinson's disease

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Parkinson's disease--a multifactorial neurodegenerative disorder.

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Alpha-synuclein in Lewy bodies

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alpha-Synuclein in filamentous inclusions of Lewy bodies from Parkinson's disease and dementia with lewy bodies

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Glial cytoplasmic inclusions in white matter oligodendrocytes of multiple system atrophy brains contain insoluble alpha-synuclein.

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Molecular cloning of cDNA encoding an unrecognized component of amyloid in Alzheimer disease

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NACP, a protein implicated in Alzheimer's disease and learning, is natively unfolded

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alpha-synuclein fibrillogenesis is nucleation-dependent. Implications for the pathogenesis of Parkinson's disease

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NACP, the precursor protein of the non-amyloid beta/A4 protein (A beta) component of Alzheimer disease amyloid, binds A beta and stimulates A beta aggregation

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Growth Factors Rescue Embryonic Dopamine Neurons from Programmed Cell Death

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Epidemiology versus genetics in Parkinson's disease: progress in resolving an age-old debate

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10.1016/S0006-8993(00)02215-0

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7 November 2019

1 reference

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7 November 2019

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