beta(2)-microglobulin induces apoptosis in HL-60 human leukemia cell line and its multidrug resistant variants overexpressing MRP1 but lacking Bax or overexpressing P-glycoprotein (Q40769236)

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English	beta(2)-microglobulin induces apoptosis in HL-60 human leukemia cell line and its multidrug resistant variants overexpressing MRP1 but lacking Bax or overexpressing P-glycoprotein	scientific article

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scholarly article

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Europe PubMed Central

23 September 2017

beta(2)-microglobulin induces apoptosis in HL-60 human leukemia cell line and its multidrug resistant variants overexpressing MRP1 but lacking Bax or overexpressing P-glycoprotein (English)

1 reference

Europe PubMed Central

23 September 2017

multiple drug resistance

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based on heuristic

inferred from title

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author name string

C H Wu

1

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Europe PubMed Central

23 September 2017

M Rastegar

2

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Europe PubMed Central

23 September 2017

J Gordon

3

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Europe PubMed Central

23 September 2017

A R Safa

4

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Europe PubMed Central

23 September 2017

language of work or name

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publication date

1 October 2001

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Europe PubMed Central

23 September 2017

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Europe PubMed Central

23 September 2017

20

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Europe PubMed Central

23 September 2017

7006-7020

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Europe PubMed Central

23 September 2017

48

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Europe PubMed Central

23 September 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1204893

0 references

Mutations of the beta2-microglobulin gene result in a lack of HLA class I molecules on melanoma cells of two patients immunized with MAGE peptides

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Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

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Structural and functional analysis of beta2 microglobulin abnormalities in human lung and breast cancer

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Mitochondrial Membrane Potential in Living Cells

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Mitochondrion as a novel target of anticancer chemotherapy

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Inhibition of caspase-3-mediated poly(ADP-ribose) polymerase (PARP) apoptotic cleavage by human PARP autoantibodies and effect on cells undergoing apoptosis.

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Characterization of beta 2-microglobulin transcripts from two teleost species

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Lack of HLA class I antigen expression by cultured melanoma cells FO-1 due to a defect in B2m gene expression

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Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ions

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Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xL.

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Megestrol acetate reverses multidrug resistance and interacts with P-glycoprotein

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Cyclosporin A binding to mitochondrial cyclophilin inhibits the permeability transition pore and protects hearts from ischaemia/reperfusion injury

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beta2-Microglobulin mutations, HLA class I antigen loss, and tumor progression in melanoma.

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Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

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Hepatocytes sensitized to tumor necrosis factor-alpha cytotoxicity undergo apoptosis through caspase-dependent and caspase-independent pathways

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Mitochondrial control of cell death

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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Mechanisms of multidrug resistance in HL60 cells. Analysis of resistance associated membrane proteins and levels of mdr gene expression

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FLICE is activated by association with the CD95 death-inducing signaling complex (DISC).

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Expression of CD95(FAS) by gene transfer does not sensitize K562 to Fas-killing

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Caspases: killer proteases

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Interactions of cyclophilin with the mitochondrial inner membrane and regulation of the permeability transition pore, and cyclosporin A-sensitive channel.

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Identification and characterization of the MDR1 promoter-enhancing factor 1 (MEF1) in the multidrug resistant HL60/VCR human acute myeloid leukemia cell line

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Molecular mechanisms of loss of beta 2-microglobulin expression in drug-resistant breast cancer sublines and its involvement in drug resistance

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Expression of the mutated p53 tumor suppressor protein and its molecular and biochemical characterization in multidrug resistant MCF-7/Adr human breast cancer cells

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Importance of poly(ADP-ribose) polymerase and its cleavage in apoptosis. Lesson from an uncleavable mutant

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Population biology of antigen presentation by MHC class I molecules

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Loss of functional beta 2-microglobulin in metastatic melanomas from five patients receiving immunotherapy

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Identification of human cancers deficient in antigen processing

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Molecular mechanisms of apoptosis induced by cytotoxic chemicals

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Human MDR 1 protein overexpression delays the apoptotic cascade in Chinese hamster ovary fibroblasts.

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Systemic injection of a tripeptide inhibits the intracellular activation of CPP32-like proteases in vivo and fully protects mice against Fas-mediated fulminant liver destruction and death

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JC-1, but not DiOC6(3) or rhodamine 123, is a reliable fluorescent probe to assess delta psi changes in intact cells: implications for studies on mitochondrial functionality during apoptosis

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Non-specific effects of methyl ketone peptide inhibitors of caspases

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Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

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Ligation of major histocompatability complex (MHC) class I molecules on human T cells induces cell death through PI-3 kinase-induced c-Jun NH2-terminal kinase activity: a novel apoptotic pathway distinct from Fas-induced apoptosis

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Executioner caspase-3, -6, and -7 perform distinct, non-redundant roles during the demolition phase of apoptosis.

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Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner

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Apoptosis signaling

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Two distinct pathways leading to nuclear apoptosis

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Bcl-2 inhibits the mitochondrial release of an apoptogenic protease

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10.1038/SJ.ONC.1204893

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Europe PubMed Central

23 September 2017

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Europe PubMed Central

23 September 2017

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