Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis (Q40697544)

22 September 2017

Mitochondrial dysfunction is an early indicator of doxorubicin-induced apoptosis (English)

1 reference

22 September 2017

Pattie S Green

1

1 reference

PubMed ID

12379319

22 September 2017

Christiaan Leeuwenburgh

series ordinal

2

1 reference

22 September 2017

1 October 2002

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Biochimica et Biophysica Acta

22 September 2017

1 reference

22 September 2017

1588

1 reference

22 September 2017

1

1 reference

22 September 2017

94-101

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Anthracycline cardiotoxicity and its prevention

22 September 2017

1 reference

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7 January 2021

Anthracycline-induced cardiotoxicity

1 reference

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7 January 2021

Effect of the oxidative stress induced by adriamycin on rat hepatocyte bioenergetics during ageing

1 reference

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7 January 2021

An electron paramagnetic resonance study of the interactions between the adriamycin semiquinone, hydrogen peroxide, iron-chelators, and radical scavengers

1 reference

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7 January 2021

Doxorubicin-induced apoptosis in endothelial cells and cardiomyocytes is ameliorated by nitrone spin traps and ebselen. Role of reactive oxygen and nitrogen species

1 reference

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7 January 2021

The exogenous NADH dehydrogenase of heart mitochondria is the key enzyme responsible for selective cardiotoxicity of anthracyclines.

1 reference

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7 January 2021

A novel superoxide radical generator in heart mitochondria

1 reference

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7 January 2021

Doxorubicin-induced cardiomyopathy

1 reference

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7 January 2021

Overexpression of metallothionein in the heart of transgenic mice suppresses doxorubicin cardiotoxicity

1 reference

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7 January 2021

Suppression of doxorubicin cardiotoxicity by overexpression of catalase in the heart of transgenic mice

1 reference

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7 January 2021

The protective role of manganese superoxide dismutase against adriamycin-induced acute cardiac toxicity in transgenic mice

1 reference

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7 January 2021

Demonstration of the existence of an organo-specific NADH dehydrogenase in heart mitochondria

1 reference

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7 January 2021

The central executioners of apoptosis: caspases or mitochondria?

1 reference

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7 January 2021

Cell suicide in health and disease

1 reference

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7 January 2021

Mitochondria-dependent apoptosis and cellular pH regulation

1 reference

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7 January 2021

Hydroxyl radical generation during exercise increases mitochondrial protein oxidation and levels of urinary dityrosine

1 reference

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7 January 2021

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

1 reference

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7 January 2021

Doxorubicin and mechanical performance of cardiac trabeculae after acute and chronic treatment: a review

1 reference

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7 January 2021

Role of apoptosis and apoptosis-related genes in cellular response and antitumor efficacy of anthracyclines.

1 reference

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7 January 2021

Doxorubicin-induced acute changes in cytotoxic aldehydes, antioxidant status and cardiac function in the rat

1 reference

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7 January 2021

An electron spin resonance study of the reduction of peroxides by anthracycline semiquinones

1 reference

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7 January 2021

Apoptosis in isolated adult cardiomyocytes exposed to adriamycin

1 reference

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7 January 2021

Apoptosis in rat cardiac myocytes induced by Fas ligand: priming for Fas-mediated apoptosis with doxorubicin

1 reference

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7 January 2021

Augmented adriamycin sensitivity in cells transduced with an antisense tumor necrosis factor gene is mediated by caspase-3 downstream from reactive oxygen species

1 reference

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7 January 2021

Complexity in simplicity: monogenic disorders and complex cardiomyopathies

1 reference

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7 January 2021

ARC, an inhibitor of apoptosis expressed in skeletal muscle and heart that interacts selectively with caspases

1 reference

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7 January 2021

ARC inhibits cytochrome c release from mitochondria and protects against hypoxia-induced apoptosis in heart-derived H9c2 cells

1 reference

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7 January 2021

The apoptotic regulatory protein ARC (apoptosis repressor with caspase recruitment domain) prevents oxidant stress-mediated cell death by preserving mitochondrial function

1 reference

https://api.crossref.org/works/10.1016%2FS0925-4439%2802%2900144-8

7 January 2021

10.1016/S0925-4439(02)00144-8

Identifiers

DOI

1 reference

release_67hwvgjsrje77e2pn4iha66psi

22 September 2017

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/67hwvgjsrje77e2pn4iha66psi

24 November 2022

mapped directly with Wikidata item

PubMed ID

12379319

1 reference

PubMed ID

12379319