Down-regulation of human papillomavirus E6/E7 oncogene by arsenic trioxide in cervical carcinoma cells (Q40690286)

22 September 2017

title

Down-regulation of human papillomavirus E6/E7 oncogene by arsenic trioxide in cervical carcinoma cells (English)

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22 September 2017

Soo-Jong Um

1

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22 September 2017

So-Young Lee

2

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22 September 2017

Eun-Joo Kim

3

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22 September 2017

Jin Myoung

4

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22 September 2017

Sung-Eun Namkoong

5

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22 September 2017

Jong-Sup Park

6

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22 September 2017

language of work or name

English

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publication date

1 July 2002

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22 September 2017

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22 September 2017

volume

181

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22 September 2017

issue

1

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22 September 2017

page(s)

11-22

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Localization of the E6-AP regions that direct human papillomavirus E6 binding, association with p53, and ubiquitination of associated proteins

22 September 2017

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Complete remission after treatment of acute promyelocytic leukemia with arsenic trioxide

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Arsenic induces apoptosis in B-cell leukaemic cell lines in vitro: activation of caspases and down-regulation of Bcl-2 protein

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Apoptosis and growth inhibition in malignant lymphocytes after treatment with arsenic trioxide at clinically achievable concentrations

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7 January 2021

Arsenic trioxide induces apoptosis in neuroblastoma cell lines through the activation of caspase 3 in vitro

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7 January 2021

Arsenic trioxide induces apoptosis in human gastric cancer cells through up-regulation of p53 and activation of caspase-3.

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Arsenic trioxide induces apoptosis of HPV16 DNA-immortalized human cervical epithelial cells and selectively inhibits viral gene expression

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Antiproliferative effects of retinoic acid/interferon in cervical carcinoma cell lines: cooperative growth suppression of IRF-1 and p53

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Functional inactivation of p73, a homolog of p53 tumor suppressor protein, by human papillomavirus E6 proteins

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Correlation of poly(ADP-ribose)polymerase andp53 expression levels in high-grade lymphomas

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Stromelysin-3 induction and interstitial collagenase repression by retinoic acid. Therapeutical implication of receptor-selective retinoids dissociating transactivation and AP-1-mediated transrepression

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Clinical trials of arsenic trioxide in hematologic and solid tumors: overview of the National Cancer Institute Cooperative Research and Development Studies

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WAF1, a potential mediator of p53 tumor suppression

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The enhancer of human papillomavirus type 16: binding sites for the ubiquitous transcription factors oct-1, NFA, TEF-2, NF1, and AP-1 participate in epithelial cell-specific transcription

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Opposite effect of NF-kB and c-Jun N-terminal kinase on p53-independent GADD45 induction by arsenite

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AP-1 transcriptional activity is regulated by a direct association between thioredoxin and Ref-1

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Inhibition of human papillomavirus-16 long control region activity by interferon-gamma overcome by p300 overexpression

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Angiogenesis Modulators Expression in Culture Cell Lines Positives for HPV-16 Oncoproteins

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7 January 2021

10.1016/S0304-3835(02)00039-3

Identifiers

DOI

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22 September 2017

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