Insulin-like growth factor-binding protein-3 potentiates epidermal growth factor action in MCF-10A mammary epithelial cells. Involvement of p44/42 and p38 mitogen-activated protein kinases (Q40689818)

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scientific article published on 13 November 2002

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English	Insulin-like growth factor-binding protein-3 potentiates epidermal growth factor action in MCF-10A mammary epithelial cells. Involvement of p44/42 and p38 mitogen-activated protein kinases	scientific article published on 13 November 2002

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scholarly article

1 reference

Europe PubMed Central

22 September 2017

Insulin-like growth factor-binding protein-3 potentiates epidermal growth factor action in MCF-10A mammary epithelial cells. Involvement of p44/42 and p38 mitogen-activated protein kinases (English)

1 reference

Europe PubMed Central

22 September 2017

author name string

Janet L Martin

1

1 reference

Europe PubMed Central

22 September 2017

Sarah M Weenink

2

1 reference

Europe PubMed Central

22 September 2017

Robert C Baxter

3

1 reference

Europe PubMed Central

22 September 2017

language of work or name

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publication date

13 November 2002

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Europe PubMed Central

22 September 2017

Journal of Biological Chemistry

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Europe PubMed Central

22 September 2017

278

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Europe PubMed Central

22 September 2017

5

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Europe PubMed Central

22 September 2017

2969-2976

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Europe PubMed Central

22 September 2017

Insulin-like growth factors and their binding proteins: biological actions

1 reference

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Retinoid modulation of insulin-like growth factor-binding proteins and inhibition of breast carcinoma proliferation

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Transforming growth factor-beta-induced cell growth inhibition in human breast cancer cells is mediated through insulin-like growth factor-binding protein-3 action.

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Insulin-like growth factor-binding protein-3 modulates expression of Bax and Bcl-2 and potentiates p53-independent radiation-induced apoptosis in human breast cancer cells

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Differential IGF-independent effects of insulin-like growth factor binding proteins (1-6) on apoptosis of breast epithelial cells.

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Insulin-like growth factor-binding protein enhancement of insulin-like growth factor-I (IGF-I)-mediated DNA synthesis and IGF-I binding in a human breast carcinoma cell line

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Insulin-like growth factor binding protein-3 is regulated by dihydrotestosterone and stimulates deoxyribonucleic acid synthesis and cell proliferation in LNCaP prostate carcinoma cells

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Development of resistance to insulin-like growth factor binding protein-3 in transfected T47D breast cancer cells.

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Associations between insulin-like growth factors and their binding proteins and other prognostic indicators in breast cancer

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Insulin-like growth factor-binding protein-3 and breast cancer survival

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Oncogenic ras causes resistance to the growth inhibitor insulin-like growth factor binding protein-3 (IGFBP-3) in breast cancer cells.

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Mutagenesis of basic amino acids in the carboxyl-terminal region of insulin-like growth factor binding protein-5 affects acid-labile subunit binding.

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Structural determinants of ligand and cell surface binding of insulin-like growth factor-binding protein-3

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Insulin-like growth factor-binding protein (IGFBP)-3 and IGFBP-5 share a common nuclear transport pathway in T47D human breast carcinoma cells.

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Antiproliferative effects of insulin-like growth factor-binding protein-3 in mesenchymal chondrogenic cell line RCJ3.1C5.18. relationship to differentiation stage

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Glycosylation of Insulin-Like Growth Factor Binding Protein-3 (IGFBP-3) is not Required for Potentiation IGF-I Action: Evidence for Processing of Cell-Bound IGFBP-3*

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Molecular mechanisms underlying ErbB2/HER2 action in breast cancer

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Regulation of epidermal growth factor receptor signaling by phosphorylation of the ras exchange factor hSOS1.

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Insulin-like growth factor-binding protein-3 activates a phosphotyrosine phosphatase. Effects on the insulin-like growth factor signaling pathway

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21 January 2018

Convergence of progesterone and epidermal growth factor signaling in breast cancer. Potentiation of mitogen-activated protein kinase pathways

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Growth inhibition by insulin-like growth factor-binding protein-3 in T47D breast cancer cells requires transforming growth factor-beta (TGF-beta ) and the type II TGF-beta receptor

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Signaling through the Smad pathway by insulin-like growth factor-binding protein-3 in breast cancer cells. Relationship to transforming growth factor-beta 1 signaling.

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21 January 2018

Direct functional interactions between insulin-like growth factor-binding protein-3 and retinoid X receptor-alpha regulate transcriptional signaling and apoptosis

1 reference

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21 January 2018

IGF-binding protein-3-induced growth inhibition and apoptosis do not require cell surface binding and nuclear translocation in human breast cancer cells

1 reference

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21 January 2018

The 16-kDa Proteolytic Fragment of Insulin-Like Growth Factor (IGF) Binding Protein-3 Inhibits the Mitogenic Action of Fibroblast Growth Factor on Mouse Fibroblasts with a Targeted Disruption of the Type 1 IGF Receptor Gene

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21 January 2018

Identifiers

10.1074/JBC.M210739200

1 reference

Europe PubMed Central

22 September 2017

1 reference

Europe PubMed Central

22 September 2017

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