Regulation of Bcl-2 during androgen-unresponsive progression of prostate cancer (Q40681607)

22 September 2017

title

Regulation of Bcl-2 during androgen-unresponsive progression of prostate cancer (English)

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22 September 2017

Rothermund CA

1

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22 September 2017

Kondrikov D

series ordinal

2

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22 September 2017

Lin MF

series ordinal

3

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22 September 2017

Vishwanatha JK

series ordinal

4

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22 September 2017

publication date

1 January 2002

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Prostate Cancer and Prostatic Diseases

22 September 2017

published in

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22 September 2017

volume

5

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22 September 2017

issue

3

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22 September 2017

page(s)

236-245

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https://scigraph.springernature.com/pub.10.1038/sj.pcan.4500582

22 September 2017

exact match

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21 January 2018

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Unusual specificity of the androgen receptor in the human prostate tumor cell line LNCaP: high affinity for progestagenic and estrogenic steroids

21 January 2018

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Activation of Rb and decline in androgen receptor protein precede retinoic acid-induced apoptosis in androgen-dependent LNCaP cells and their androgen-independent derivative

21 January 2018

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Posttranslational modification of Bcl-2 facilitates its proteasome-dependent degradation: molecular characterization of the involved signaling pathway.

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Deletion of the loop region of Bcl-2 completely blocks paclitaxel-induced apoptosis

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BCL-2 family members and the mitochondria in apoptosis

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7 January 2021

Overview of phase III trials on combined androgen treatment in patients with metastatic prostate cancer

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Resistance to apoptosis and up regulation of Bcl-2 in benign prostatic hyperplasia after androgen deprivation

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Expression of human prostatic acid phosphatase correlates with androgen-stimulated cell proliferation in prostate cancer cell lines

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Identifiers

DOI

10.1038/SJ.PCAN.4500582

1 reference

Dimensions Publication ID

22 September 2017

1040133574

0 references

1 reference