A critical role of glutathione in determining apoptosis sensitivity and resistance in leukemia cells (Q40563388)

21 September 2017

A critical role of glutathione in determining apoptosis sensitivity and resistance in leukemia cells (English)

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21 September 2017

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Klaus-Michael Debatin

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3

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21 September 2017

Friesen C

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21 September 2017

Kiess Y

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2

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21 September 2017

language of work or name

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1 July 2004

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Cell Death & Differentiation

21 September 2017

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21 September 2017

11 Suppl 1

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21 September 2017

S73-85

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https://scigraph.springernature.com/pub.10.1038/sj.cdd.4401431

21 September 2017

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Involvement of the CD95 (APO–1/Fas) receptor/ligand system in drug–induced apoptosis in leukemia cells

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Deficient activation of the CD95 (APO-1/Fas) system in drug-resistant cells

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Induction of CD95 ligand and apoptosis by doxorubicin is modulated by the redox state in chemosensitive- and drug-resistant tumor cells

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Drug-induced apoptosis in hepatoma cells is mediated by the CD95 (APO-1/Fas) receptor/ligand system and involves activation of wild-type p53.

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DNA damaging agents induce expression of Fas ligand and subsequent apoptosis in T lymphocytes via the activation of NF-kappa B and AP-1.

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

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Molecular characterization of mitochondrial apoptosis-inducing factor

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Apoptosis inducing factor (AIF): a phylogenetically old, caspase-independent effector of cell death

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Mitochondrial release of caspase-2 and -9 during the apoptotic process

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An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

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A conserved XIAP-interaction motif in caspase-9 and Smac/DIABLO regulates caspase activity and apoptosis

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Cleavage of human inhibitor of apoptosis protein XIAP results in fragments with distinct specificities for caspases

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Mitochondrial permeability transition is a central coordinating event of apoptosis

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Two CD95 (APO-1/Fas) signaling pathways

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Cell type specific involvement of death receptor and mitochondrial pathways in drug-induced apoptosis

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The role of oxygen-derived free radicals in the cytotoxicity of doxorubicin in multidrug resistant and sensitive human ovarian cancer cells

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Glutathione dependence of caspase-8 activation at the death-inducing signaling complex

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Monoclonal antibody-mediated tumor regression by induction of apoptosis.

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A rapid and simple method for measuring thymocyte apoptosis by propidium iodide staining and flow cytometry

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Oxidants in mitochondria: from physiology to diseases

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Cell fusion studies to examine the mechanism for etoposide resistance in Chinese hamster V79 spheroids

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Thiol-mediated inhibition of FAS and CD2 apoptotic signaling in activated human peripheral T cells

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Chemotherapy-induced apoptosis

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7 January 2021

Chemosensitivity of solid tumor cellsin vitro is related to activation of the CD95 system

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7 January 2021

Arsenic trioxide, a novel mitochondriotoxic anticancer agent?

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7 January 2021

Cleavage of BID during cytotoxic drug and UV radiation-induced apoptosis occurs downstream of the point of Bcl-2 action and is catalysed by caspase-3: a potential feedback loop for amplification of apoptosis-associated mitochondrial cytochrome c rel

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7 January 2021

Fas-mediated apoptosis is modulated by intracellular glutathione in human T cells

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7 January 2021

Chapter 3 Glutathione and Cellular Resistance to Anti-Cancer Drugs

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7 January 2021

Reactive oxygen intermediates are involved in the induction of CD95 ligand mRNA expression by cytostatic drugs in hepatoma cells

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7 January 2021

Inhibition of activation-induced death in T cell hybridomas by thiol antioxidants: oxidative stress as a mediator of apoptosis.

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7 January 2021

Cellular pharmacokinetics of doxorubicin in patients with chronic lymphocytic leukemia: comparison of bolus administration and continuous infusion

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401431

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Dimensions Publication ID

21 September 2017

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