Oxidative stress attenuates Fas-mediated apoptosis in Jurkat T cell line through Bfl-1 induction. (Q40481699)

20 September 2017

Oxidative stress attenuates Fas-mediated apoptosis in Jurkat T cell line through Bfl-1 induction. (English)

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20 September 2017

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Heejung Kim

1

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20 September 2017

Yong-Nyun Kim

series ordinal

2

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20 September 2017

Hyungsoo Kim

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3

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20 September 2017

Chul-Woo Kim

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4

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20 September 2017

language of work or name

English

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publication date

1 February 2005

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20 September 2017

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20 September 2017

24

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20 September 2017

1252-1261

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20 September 2017

7

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1208282

20 September 2017

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Blocking NF-kappaB activation in Jurkat leukemic T cells converts the survival agent and tumor promoter PMA into an apoptotic effector.

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Hydrogen peroxide-mediated phosphorylation of ERK1/2, Akt/PKB and JNK in cortical neurones: dependence on Ca(2+) and PI3-kinase

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Hydrogen peroxide induces upregulation of Fas in human airway epithelial cells via the activation of PARP-p53 pathway

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Enhancement of T cell receptor signaling by a mild oxidative shift in the intracellular thiol pool

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Apoptosis or growth arrest: modulation of the cellular response to p53 by proliferative signals

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Molecular mechanisms of activated T cell death in vivo

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Redox regulation of cellular signalling

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Suppression of NF kappa B activation and NF kappa B-dependent gene expression by tepoxalin, a dual inhibitor of cyclooxygenase and 5-lipoxygenase

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NF-kappaB-mediated up-regulation of Bcl-x and Bfl-1/A1 is required for CD40 survival signaling in B lymphocytes

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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Inhibition of activated/memory (CD45RO(+)) T cells by oxidative stress associated with block of NF-kappaB activation

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Human A1, a Bcl-2-related gene, is induced in leukemic cells by cytokines as well as differentiating factors

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Human A1 expression in acute myeloid leukemia and its relationship to Bcl-2 expression

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Oxidative stress by tumor-derived macrophages suppresses the expression of CD3 zeta chain of T-cell receptor complex and antigen-specific T-cell responses

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JNK1 activity lowers the cellular production of H2O2 and modulates the growth arrest response to scavenging of H2O2 by catalase

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Oxidative stress-mediated down-regulation of bcl-2 promoter in hippocampal neurons.

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Inhibition of NF-kappa B activation in human T-cell lines by anetholdithiolthione

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Bcl-2 family member Bfl-1/A1 sequesters truncated bid to inhibit is collaboration with pro-apoptotic Bak or Bax

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Structural basis of BFL-1 for its interaction with BAX and its anti-apoptotic action in mammalian and yeast cells

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Identifiers

DOI

10.1038/SJ.ONC.1208282

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Dimensions Publication ID

20 September 2017

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