Cytokine cooperation in renal tubular cell injury: the role of TWEAK. (Q40227178)

18 September 2017

Cytokine cooperation in renal tubular cell injury: the role of TWEAK. (English)

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18 September 2017

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Justo P

1

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18 September 2017

Sanz AB

2

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18 September 2017

Sanchez-Niño MD

3

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18 September 2017

Winkles JA

4

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18 September 2017

Lorz C

5

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18 September 2017

Egido J

6

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18 September 2017

Ortiz A

7

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18 September 2017

language of work or name

English

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publication date

27 September 2006

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18 September 2017

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18 September 2017

70

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18 September 2017

1750-1758

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18 September 2017

10

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https://scigraph.springernature.com/pub.10.1038/sj.ki.5001866

18 September 2017

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TNF-alpha mediates obstruction-induced renal tubular cell apoptosis and proapoptotic signaling

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TWEAK, a new secreted ligand in the tumor necrosis factor family that weakly induces apoptosis

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TWEAK, a member of the TNF superfamily, is a multifunctional cytokine that binds the TweakR/Fn14 receptor

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The role of TWEAK/Fn14 in the pathogenesis of inflammation and systemic autoimmunity

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Proinflammatory activity of TWEAK on human dermal fibroblasts and synoviocytes: blocking and enhancing effects of anti-TWEAK monoclonal antibodies

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TWEAK induces angiogenesis and proliferation of endothelial cells

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A novel TNF receptor family member binds TWEAK and is implicated in angiogenesis

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The mitogen-inducible Fn14 gene encodes a type I transmembrane protein that modulates fibroblast adhesion and migration

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Fibroblast growth factor-inducible 14 mediates multiple pathways of TWEAK-induced cell death

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Expression of Smac/Diablo in tubular epithelial cells and during acute renal failure.

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Caspases: enemies within

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

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The apoptotic ligands TRAIL, TWEAK, and Fas ligand mediate monocyte death induced by autologous lupus T cells

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Multiple pathways of TWEAK-induced cell death

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Proinflammatory effects of TWEAK/Fn14 interactions in glomerular mesangial cells

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The Fn14 cytoplasmic tail binds tumour-necrosis-factor-receptor-associated factors 1, 2, 3 and 5 and mediates nuclear factor-kappaB activation

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TWEAK mediates signal transduction and differentiation of RAW264.7 cells in the absence of Fn14/TweakR. Evidence for a second TWEAK receptor

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Characterization of a renal tubular epithelial cell line which secretes the autologous target antigen of autoimmune experimental interstitial nephritis

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7 January 2021

Role of Parathyroid Hormone-Related Protein in Tubulointerstitial Apoptosis and Fibrosis after Folic Acid-Induced Nephrotoxicity

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7 January 2021

Proliferation of bone marrow-derived cells contributes to regeneration after folic acid-induced acute tubular injury.

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7 January 2021

Attenuation of folic acid-induced renal inflammatory injury in platelet-activating factor receptor-deficient mice

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7 January 2021

Blocking tumor necrosis factor-alpha inhibits folic acid-induced acute renal failure

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7 January 2021

Identifiers

DOI

10.1038/SJ.KI.5001866

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18 September 2017

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