Down regulation of TRIF, TLR3, and MAVS in HCV infected liver correlates with the outcome of infection. (Q40211217)

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19 March 2020

title

Down regulation of TRIF, TLR3, and MAVS in HCV infected liver correlates with the outcome of infection (English)

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19 March 2020

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2

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19 March 2020

author name string

Premashis Kar

1

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19 March 2020

Phani Kumar Gumma

3

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19 March 2020

Soumya Jyoti Chowdhury

4

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19 March 2020

Vijay Kumar Karra

5

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19 March 2020

publication date

4 September 2017

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Journal of Medical Virology

19 March 2020

published in

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19 March 2020

volume

89

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19 March 2020

issue

12

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19 March 2020

page(s)

2165-2172

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Global challenges in liver disease

19 March 2020

cites work

1 reference

Quantitative liver function tests improve the prediction of clinical outcomes in chronic hepatitis C: results from the Hepatitis C Antiviral Long-term Treatment Against Cirrhosis Trial

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21 January 2018

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A wide range of NS3/4A protease catalytic efficiencies in HCV-infected individuals

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21 January 2018

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MDA5 plays a critical role in interferon response during hepatitis C virus infection

21 January 2018

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Activation of Type I and III Interferon Response by Mitochondrial and Peroxisomal MAVS and Inhibition by Hepatitis C Virus

21 January 2018

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Toll-Like Receptor 3 Mediates Establishment of an Antiviral State against Hepatitis C Virus in Hepatoma Cells

21 January 2018

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21 January 2018

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21 January 2018

1 reference

Clinical, virologic, histologic, and biochemical outcomes after successful HCV therapy: A 5-year follow-up of 150 patients

21 January 2018

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Multi-step regulation of interferon induction by hepatitis C virus.

21 January 2018

1 reference

Effect of IL28B genotype on hepatitis B and C virus infection

21 January 2018

1 reference

IL28B rs12980275 variant as a predictor of sustained virologic response to pegylated-interferon and ribavirin in chronic hepatitis C patients: A systematic review and meta-analysis.

21 January 2018

1 reference

21 January 2018

Identifiers

DOI

10.1002/JMV.24849

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19 March 2020

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