Increased apoptosis of parasympathetic but not enteric neurons in mice lacking GFRalpha2. (Q40159883)

18 September 2017

Increased apoptosis of parasympathetic but not enteric neurons in mice lacking GFRalpha2. (English)

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18 September 2017

Meri Lähteenmäki

1

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18 September 2017

Jussi Kupari

series ordinal

2

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18 September 2017

Matti S Airaksinen

series ordinal

3

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18 September 2017

language of work or name

English

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publication date

21 February 2007

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18 September 2017

8

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based on heuristic

inferred from page(s)

published in

Developmental Biology

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18 September 2017

305

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18 September 2017

1

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18 September 2017

325-332

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Estimation of nuclear population from microtome sections

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NT-3, like NGF, is required for survival of sympathetic neurons, but not their precursors.

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GDNF availability determines enteric neuron number by controlling precursor proliferation.

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Gene targeting reveals a critical role for neurturin in the development and maintenance of enteric, sensory, and parasympathetic neurons

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GDNF family receptors in the embryonic and postnatal rat heart and reduced cholinergic innervation in mice hearts lacking ret or GFRalpha2.

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Retarded growth and deficits in the enteric and parasympathetic nervous system in mice lacking GFR alpha2, a functional neurturin receptor.

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Alimentary tract innervation deficits and dysfunction in mice lacking GDNF family receptor alpha2.

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Parasympathetic innervation and function of endocrine pancreas requires the glial cell line-derived factor family receptor alpha2 (GFRalpha2).

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Assessment of protein gene product 9.5 as a marker of neural crest-derived precursor cells in the developing enteric nervous system.

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In situ immunodetection of activated caspase-3 in apoptotic neurons in the developing nervous system

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Region of caspase-3 activation and programmed cell death in the early development of the mouse forebrain

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Neurturin signalling via GFRalpha2 is essential for innervation of glandular but not muscle targets of sacral parasympathetic ganglion neurons

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Widespread elimination of naturally occurring neuronal death in Bax-deficient mice

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The location and phenotype of proliferating neural-crest-derived cells in the developing mouse gut.

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Diversity in the mechanisms of neuronal cell death.

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Distinct roles for GFRalpha1 and GFRalpha2 signalling in different cranial parasympathetic ganglia in vivo

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inferred from DOI database lookup

7 January 2021

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Identifiers

DOI

10.1016/J.YDBIO.2007.02.016

1 reference

18 September 2017

1 reference