Loss of FANCC function is associated with failure to inhibit late firing replication origins after DNA cross-linking (Q40134910)

17 September 2017

Loss of FANCC function is associated with failure to inhibit late firing replication origins after DNA cross-linking (English)

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17 September 2017

3

David M Hockenbery

1 reference

17 September 2017

Randall A Phelps

1

1 reference

PubMed ID

17490643

17 September 2017

Helene Gingras

series ordinal

2

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PubMed ID

17490643

17 September 2017

language of work or name

English

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publication date

6 April 2007

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Experimental Cell Research

17 September 2017

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17 September 2017

313

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17 September 2017

11

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17 September 2017

2283-2292

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The Fanconi anemia/BRCA pathway: a coordinator of cross-link repair

17 September 2017

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Cancer in Fanconi anemia.

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G2 phase cell cycle disturbance as a manifestation of genetic cell damage

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Role of the cdc25C phosphatase in G2 arrest induced by nitrogen mustard

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Distinct patterns of cell cycle disturbance elicited by compounds interfering with DNA topoisomerase I and II activity

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Arrest of S-phase progression is impaired in Fanconi anemia cells

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Damage-resistant DNA synthesis in Fanconi anemia cells treated with a DNA cross-linking agent

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DNA replication is required To elicit cellular responses to psoralen-induced DNA interstrand cross-links.

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The 4N cell cycle delay in Fanconi anemia reflects growth arrest in late S phase

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Cell cycle arrest and DNA endoreduplication following p21Waf1/Cip1 expression

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p21 is a universal inhibitor of cyclin kinases

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Nuclear accumulation of p21Cip1 at the onset of mitosis: a role at the G2/M-phase transition

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Requirement for p53 and p21 to sustain G2 arrest after DNA damage

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Cell cycle control of DNA replication

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p53 accumulates but is functionally impaired when DNA synthesis is blocked

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A role for the Fanconi anemia C protein in maintaining the DNA damage-induced G2 checkpoint

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Fanconi anemia type C and p53 cooperate in apoptosis and tumorigenesis.

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An ATR- and Cdc7-dependent DNA damage checkpoint that inhibits initiation of DNA replication

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Sensing DNA damage through ATRIP recognition of RPA-ssDNA complexes

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Functional uncoupling of MCM helicase and DNA polymerase activities activates the ATR-dependent checkpoint.

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Interaction of the Fanconi anemia proteins and BRCA1 in a common pathway

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A novel ubiquitin ligase is deficient in Fanconi anemia

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ATR couples FANCD2 monoubiquitination to the DNA-damage response

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Cdc7p-Dbf4p kinase binds to chromatin during S phase and is regulated by both the APC and the RAD53 checkpoint pathway

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The DNA crosslink-induced S-phase checkpoint depends on ATR-CHK1 and ATR-NBS1-FANCD2 pathways

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Schizosaccharomyces pombe Swi1, Swi3, and Hsk1 are components of a novel S-phase response pathway to alkylation damage

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Repair of intermediate structures produced at DNA interstrand cross-links in Saccharomyces cerevisiae

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Multiple roles of Rev3, the catalytic subunit of polzeta in maintaining genome stability in vertebrates

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DNA damage-processing pathways involved in the eukaryotic cellular response to anticancer DNA cross-linking drugs

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Repair of an interstrand DNA cross-link initiated by ERCC1-XPF repair/recombination nuclease.

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Phosphorylation of XPB helicase regulates TFIIH nucleotide excision repair activity

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Repair of DNA interstrand cross-links

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DNA repair defects channel interstrand DNA cross-links into alternate recombinational and error-prone repair pathways

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Elevated homologous recombination activity in fanconi anemia fibroblasts

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ATR and ATM regulate the timing of DNA replication origin firing

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Control of replication origin density and firing time in Xenopus egg extracts: role of a caffeine-sensitive, ATR-dependent checkpoint.

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Saccharomyces cerevisiae Rrm3p DNA helicase promotes genome integrity by preventing replication fork stalling: viability of rrm3 cells requires the intra-S-phase checkpoint and fork restart activities

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The fanconi anemia pathway requires FAA phosphorylation and FAA/FAC nuclear accumulation

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Positional cloning of a novel Fanconi anemia gene, FANCD2

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10.1016/J.YEXCR.2007.03.034

7 January 2021

Identifiers

DOI

1 reference

17 September 2017

1 reference

PubMed ID

17490643