Frequency and clinicopathologic profile of PIK3CA mutant GISTs: molecular genetic study of 529 cases. (Q40076921)

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5 March 2020

title

Frequency and clinicopathologic profile of PIK3CA mutant GISTs: molecular genetic study of 529 cases (English)

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Artur Kowalik

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Markku Miettinen

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1

Jerzy Lasota

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Stanislaw Gozdz

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Małgorzata Chłopek

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5 March 2020

Anna Felisiak-Golabek

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object named as

Anna Felisiak-Golabek

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Sebastian Zięba

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author name string

Zeng-Feng Wang

7

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Tiffany Coates

8

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Janusz Kopczynski

9

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Maarit Sarlomo-Rikala

11

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publication date

22 January 2016

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5 March 2020

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volume

29

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page(s)

275-282

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issue

3

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https://scigraph.springernature.com/pub.10.1038/modpathol.2015.160

5 March 2020

exact match

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cites work

Quadruple wild-type (WT) GIST: defining the subset of GIST that lacks abnormalities of KIT, PDGFRA, SDH, or RAS signaling pathways

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PIK3CA mutations frequently coexist with RAS and BRAF mutations in patients with advanced cancers

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KRAS and KIT Gatekeeper Mutations Confer Polyclonal Primary Imatinib Resistance in GI Stromal Tumors: Relevance of Concomitant Phosphatidylinositol 3-Kinase/AKT Dysregulation

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Prognostic Role of PIK3CA Mutation in Colorectal Cancer: Cohort Study and Literature Review

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Frequent mono-allelic loss associated with deficient PTEN expression in imatinib-resistant gastrointestinal stromal tumors

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Exploring novel therapeutic targets in GIST: focus on the PI3K/Akt/mTOR pathway

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A potent combination of the novel PI3K Inhibitor, GDC-0941, with imatinib in gastrointestinal stromal tumor xenografts: long-lasting responses after treatment withdrawal

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PIK3CA mutation impact on survival in breast cancer patients and in ERα, PR and ERBB2-based subgroups

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The activated targets of mTOR signaling pathway are characteristic for PDGFRA mutant and wild-type rather than KIT mutant GISTs.

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Dedifferentiation in gastrointestinal stromal tumor to an anaplastic KIT-negative phenotype: a diagnostic pitfall: morphologic and molecular characterization of 8 cases occurring either de novo or after imatinib therapy.

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No KRAS mutations found in gastrointestinal stromal tumors (GISTs): molecular genetic study of 514 cases

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Novel oncogene and tumor suppressor mutations in KIT and PDGFRA wild type gastrointestinal stromal tumors revealed by next generation sequencing.

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Heterogeneity of kinase inhibitor resistance mechanisms in GIST

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PIK3CA mutation H1047R is associated with response to PI3K/AKT/mTOR signaling pathway inhibitors in early-phase clinical trials

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Targeted ultra-deep sequencing reveals recurrent and mutually exclusive mutations of cancer genes in blastic plasmacytoid dendritic cell neoplasm

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PubMed

https://pubmed.ncbi.nlm.nih.gov/26796526