DJ-1 modulates UV-induced oxidative stress signaling through the suppression of MEKK1 and cell death (Q40007595)

16 September 2017

DJ-1 modulates UV-induced oxidative stress signaling through the suppression of MEKK1 and cell death (English)

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16 September 2017

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J-S Mo

1

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16 September 2017

M-Y Kim

2

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16 September 2017

E-J Ann

3

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16 September 2017

J-A Hong

4

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16 September 2017

H-S Park

5

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16 September 2017

29 February 2008

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Cell Death & Differentiation

16 September 2017

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16 September 2017

15

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16 September 2017

1030-1041

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16 September 2017

6

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https://scigraph.springernature.com/pub.10.1038/cdd.2008.26

16 September 2017

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Shedding of a rat epididymal sperm protein associated with infertility induced by ornidazole and alpha-chlorohydrin

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Mutations in the DJ-1 gene associated with autosomal recessive early-onset parkinsonism

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The role of pathogenic DJ-1 mutations in Parkinson's disease.

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DJ-1 (PARK7) mutations are less frequent than Parkin (PARK2) mutations in early-onset Parkinson disease.

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The crystal structure of DJ-1, a protein related to male fertility and Parkinson's disease

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Crystal structure of human DJ-1, a protein associated with early onset Parkinson's disease

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The 1.1-A resolution crystal structure of DJ-1, the protein mutated in autosomal recessive early onset Parkinson's disease

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DJ-1, a cancer- and Parkinson's disease-associated protein, stabilizes the antioxidant transcriptional master regulator Nrf2

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Mutational analysis of DJ-1 in Drosophila implicates functional inactivation by oxidative damage and aging

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The Parkinson's disease protein DJ-1 is neuroprotective due to cysteine-sulfinic acid-driven mitochondrial localization

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DJ-1 is an indicator for endogenous reactive oxygen species elicited by endotoxin

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Oxidized forms of peroxiredoxins and DJ-1 on two-dimensional gels increased in response to sublethal levels of paraquat

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DJ-1 has a role in antioxidative stress to prevent cell death

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Down regulation of DJ-1 enhances cell death by oxidative stress, ER stress, and proteasome inhibition

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Proper SUMO-1 conjugation is essential to DJ-1 to exert its full activities

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Differential Effects of Parkinson's Disease-associated Mutations on Stability and Folding of DJ-1

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Familial Parkinson's disease-associated L166P mutation disrupts DJ-1 protein folding and function

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L166P mutant DJ-1, causative for recessive Parkinson's disease, is degraded through the ubiquitin-proteasome system

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The DJ-1L166P mutant protein associated with early onset Parkinson's disease is unstable and forms higher-order protein complexes

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DJ-1, a novel regulator of the tumor suppressor PTEN.

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Inactivation of Drosophila DJ-1 leads to impairments of oxidative stress response and phosphatidylinositol 3-kinase/Akt signaling

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Interaction of DJ-1 with Daxx inhibits apoptosis signal-regulating kinase 1 activity and cell death

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Signal transduction by the c-Jun N-terminal kinase (JNK)--from inflammation to development

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Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis.

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Hsp72 functions as a natural inhibitory protein of c-Jun N-terminal kinase

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JNK1: a protein kinase stimulated by UV light and Ha-Ras that binds and phosphorylates the c-Jun activation domain

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Exchange of N-CoR corepressor and Tip60 coactivator complexes links gene expression by NF-kappaB and beta-amyloid precursor protein

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Activation of the IkappaB alpha kinase complex by MEKK1, a kinase of the JNK pathway

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Interaction among mitochondria, mitogen-activated protein kinases, and nuclear factor-kappaB in cellular models of Parkinson's disease

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Dopaminergic cell death induced by MPP(+), oxidant and specific neurotoxicants shares the common molecular mechanism

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Gene transfer of the JNK interacting protein-1 protects dopaminergic neurons in the MPTP model of Parkinson's disease

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Nitric oxide negatively regulates c-Jun N-terminal kinase/stress-activated protein kinase by means of S-nitrosylation

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Early-onset Parkinson's disease caused by a compound heterozygous DJ-1 mutation

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Genetics of Parkinson's disease: what do mutations in DJ-1 tell us?

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7 January 2021

Detection of cathepsin B up-regulation in neoplastic thyroid tissues by proteomic analysis

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7 January 2021

A missense mutation (L166P) in DJ-1, linked to familial Parkinson's disease, confers reduced protein stability and impairs homo-oligomerization.

1 reference

https://api.crossref.org/works/10.1038%2FCDD.2008.26

7 January 2021

MPTP activates c-Jun NH(2)-terminal kinase (JNK) and its upstream regulatory kinase MKK4 in nigrostriatal neurons in vivo

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7 January 2021

Identifiers

DOI

10.1038/CDD.2008.26

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Dimensions Publication ID

16 September 2017

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