Molecular mechanism of Zn2+ agonism in the extracellular domain of GPR39. (Q39966942)

16 September 2017

Molecular mechanism of Zn2+ agonism in the extracellular domain of GPR39. (English)

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16 September 2017

Birgitte Holst

2

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Thue W Schwartz

object named as

Thue W Schwartz

series ordinal

3

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author name string

Laura Storjohann

series ordinal

1

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PubMed ID

18588883

16 September 2017

language of work or name

English

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publication date

25 June 2008

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16 September 2017

6

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inferred from page(s)

published in

FEBS Letters

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16 September 2017

582

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16 September 2017

17

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16 September 2017

2583-2588

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The neurobiology of zinc in health and disease

16 September 2017

1 reference

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Metal ion effects on ion channel gating

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Refinement of a homology model of the mu-opioid receptor using distance constraints from intrinsic and engineered zinc-binding sites

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Identification of a Zn2+-binding site on the dopamine D2 receptor.

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Metal ion-mediated agonism and agonist enhancement in melanocortin MC1 and MC4 receptors

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Allosteric modulation of beta2-adrenergic receptor by Zn(2+).

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Identification of an allosteric binding site for Zn2+ on the beta2 adrenergic receptor

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GPR39 splice variants versus antisense gene LYPD1: expression and regulation in gastrointestinal tract, endocrine pancreas, liver, and white adipose tissue

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1 reference

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GPR39 signaling is stimulated by zinc ions but not by obestatin

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Obestatin, a peptide encoded by the ghrelin gene, opposes ghrelin's effects on food intake

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Obestatin does not activate orphan G protein-coupled receptor GPR39

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Normal food intake and body weight in mice lacking the G protein-coupled receptor GPR39.

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Isolation of Zn2+ as an endogenous agonist of GPR39 from fetal bovine serum

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Engineering hybrid genes without the use of restriction enzymes: gene splicing by overlap extension

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Metal ion site engineering indicates a global toggle switch model for seven-transmembrane receptor activation.

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High constitutive signaling of the ghrelin receptor--identification of a potent inverse agonist

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Nonpeptide and peptide growth hormone secretagogues act both as ghrelin receptor agonist and as positive or negative allosteric modulators of ghrelin signaling

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Loss of constitutive activity of the growth hormone secretagogue receptor in familial short stature

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Regulation of serine protease activity by an engineered metal switch

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Kinetic aspects of compartmental storage and secretion of insulin and zinc

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Islet beta-cell secretion determines glucagon release from neighbouring alpha-cells.

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Zinc, the pancreas, and diabetes: insights from rodent studies and future directions

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A genome-wide association study identifies novel risk loci for type 2 diabetes

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The cation efflux transporter ZnT8 (Slc30A8) is a major autoantigen in human type 1 diabetes

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1 reference

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Role of cellular zinc in programmed cell death: temporal relationship between zinc depletion, activation of caspases, and cleavage of Sp family transcription factors.

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The constitutively active orphan G-protein-coupled receptor GPR39 protects from cell death by increasing secretion of pigment epithelium-derived growth factor

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1 reference

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10.1016/J.FEBSLET.2008.06.030

21 January 2018

Identifiers

DOI

1 reference

16 September 2017

1 reference

PubMed ID

18588883