Silencing of elongation factor-2 kinase potentiates the effect of 2-deoxy-D-glucose against human glioma cells through blunting of autophagy (Q39879662)

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English	Silencing of elongation factor-2 kinase potentiates the effect of 2-deoxy-D-glucose against human glioma cells through blunting of autophagy	scientific article

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scholarly article

1 reference

Europe PubMed Central

15 September 2017

Silencing of elongation factor-2 kinase potentiates the effect of 2-deoxy-D-glucose against human glioma cells through blunting of autophagy (English)

1 reference

Europe PubMed Central

15 September 2017

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author name string

Hao Wu

1

1 reference

Europe PubMed Central

15 September 2017

Hua Zhu

2

1 reference

Europe PubMed Central

15 September 2017

David X Liu

3

1 reference

Europe PubMed Central

15 September 2017

Ting-Kuang Niu

4

1 reference

Europe PubMed Central

15 September 2017

Xingcong Ren

5

1 reference

Europe PubMed Central

15 September 2017

Rajesh Patel

6

1 reference

Europe PubMed Central

15 September 2017

William N Hait

7

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Europe PubMed Central

15 September 2017

Jin-Ming Yang

8

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Europe PubMed Central

15 September 2017

language of work or name

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publication date

24 February 2009

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Europe PubMed Central

15 September 2017

Cancer Research

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Europe PubMed Central

15 September 2017

69

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Europe PubMed Central

15 September 2017

6

1 reference

Europe PubMed Central

15 September 2017

2453-2460

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Europe PubMed Central

15 September 2017

DAP-kinase is a mediator of endoplasmic reticulum stress-induced caspase activation and autophagic cell death.

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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p53 regulates mitochondrial respiration

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Elongation factor-2 kinase regulates autophagy in human glioblastoma cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2907516

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Inhibition of glutamate cysteine ligase activity sensitizes human breast cancer cells to the toxicity of 2-deoxy-D-glucose.

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Lysosomes and autophagy in cell death control

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2-deoxy-D-glucose enhances the cytotoxicity of topoisomerase inhibitors in human tumor cell lines.

1 reference

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Stimulation of the AMP-activated protein kinase leads to activation of eukaryotic elongation factor 2 kinase and to its phosphorylation at a novel site, serine 398

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Identification and characterization of an inhibitor of eukaryotic elongation factor 2 kinase against human cancer cell lines

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On the Origin of Cancer Cells

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Activation of AMP-activated protein kinase leads to the phosphorylation of elongation factor 2 and an inhibition of protein synthesis

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Regulation of elongation factor 2 kinase by p90(RSK1) and p70 S6 kinase.

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Activity and regulation by growth factors of calmodulin-dependent protein kinase III (elongation factor 2-kinase) in human breast cancer

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Identification of a new class of protein kinases represented by eukaryotic elongation factor-2 kinase

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Role of calmodulin-dependent phosphorylation of elongation factor 2 in the proliferation of rat glial cells

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2-Deoxy-D-glucose combined with cisplatin enhances cytotoxicity via metabolic oxidative stress in human head and neck cancer cells

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Phosphorylation of elongation factor 2 in normal and malignant rat glial cells

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Question 7: optimized energy consumption for protein synthesis.

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Effect of 2-deoxy-D-glucose on various malignant cell lines in vitro.

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2-Deoxyglucose as an energy restriction mimetic agent: effects on mammary carcinogenesis and on mammary tumor cell growth in vitro

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2-Deoxy-D-glucose-induced cytotoxicity and radiosensitization in tumor cells is mediated via disruptions in thiol metabolism.

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Ca2+/calmodulin-dependent phosphorylation of elongation factor 2

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12 December 2020

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10.1158/0008-5472.CAN-08-2872

1 reference

Europe PubMed Central

15 September 2017

1 reference

Europe PubMed Central

15 September 2017

1 reference

Europe PubMed Central

15 September 2017

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