A Japanese encephalitis virus vaccine candidate strain is attenuated by decreasing its interferon antagonistic ability (Q39861100)

15 September 2017

A Japanese encephalitis virus vaccine candidate strain is attenuated by decreasing its interferon antagonistic ability (English)

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Japanese encephalitis virus

15 September 2017

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Japanese encephalitis

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Yi-Ling Lin

5

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author name string

Jian-Jong Liang

1

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15 September 2017

Ching-Len Liao

2

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15 September 2017

Jia-Teh Liao

3

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15 September 2017

Yi-Ling Lee

4

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15 September 2017

language of work or name

English

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publication date

13 March 2009

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15 September 2017

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15 September 2017

27

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15 September 2017

21

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15 September 2017

2746-2754

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New initiatives for the control of Japanese encephalitis by vaccination: minutes of a WHO/CVI meeting, Bangkok, Thailand, 13-15 October 1998.

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Recent advances in Japanese encephalitis.

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Immunopathology of flavivirus infections

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Innate and Adaptive Immune Responses Determine Protection against Disseminated Infection by West Nile Encephalitis Virus

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B cells and antibody play critical roles in the immediate defense of disseminated infection by West Nile encephalitis virus.

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A Critical Role for Induced IgM in the Protection against West Nile Virus Infection

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CD8+ T cells mediate recovery and immunopathology in West Nile virus encephalitis

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The host response to West Nile Virus infection limits viral spread through the activation of the interferon regulatory factor 3 pathway

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West Nile virus evades activation of interferon regulatory factor 3 through RIG-I-dependent and -independent pathways without antagonizing host defense signaling

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1 reference

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Flavivirus induces interferon-beta gene expression through a pathway involving RIG-I-dependent IRF-3 and PI3K-dependent NF-kappaB activation

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Interferons at age 50: past, current and future impact on biomedicine

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Role of type I and type II interferon responses in recovery from infection with an encephalitic flavivirus

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Alpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survival

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Inverse interference: how viruses fight the interferon system

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Blocking of the alpha interferon-induced Jak-Stat signaling pathway by Japanese encephalitis virus infection

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1 reference

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Blocking of Interferon-Induced Jak-Stat Signaling by Japanese Encephalitis Virus NS5 through a Protein Tyrosine Phosphatase-Mediated Mechanism

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West Nile virus inhibits the signal transduction pathway of alpha interferon

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Inhibition of interferon signaling by the New York 99 strain and Kunjin subtype of West Nile virus involves blockage of STAT1 and STAT2 activation by nonstructural proteins

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Resistance to Alpha/Beta Interferon Is a Determinant of West Nile Virus Replication Fitness and Virulence

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1 reference

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Generation and characterization of organ-tropism mutants of Japanese encephalitis virus in vivo and in vitro

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A highly attenuated strain of Japanese encephalitis virus induces a protective immune response in mice

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Targeted disruption of the mouse Stat1 gene results in compromised innate immunity to viral disease

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Self-cleavage of hepatitis delta virus genomic strand RNA is enhanced under partially denaturing conditions

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Self-cleavage of a 71 nucleotide-long ribozyme derived from hepatitis delta virus genomic RNA

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Infectious RNA transcribed from stably cloned full-length cDNA of dengue type 4 virus

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Generation of deletion and point mutations with one primer in a single cloning step

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Beta 2-microglobulin-, CD8+ T-cell-deficient mice survive inoculation with high doses of vaccinia virus and exhibit altered IgG responses

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Mechanisms of type-I- and type-II-interferon-mediated signalling

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Highly sulfated forms of heparin sulfate are involved in japanese encephalitis virus infection.

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Interferon inhibits dengue virus infection by preventing translation of viral RNA through a PKR-independent mechanism

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Modulation of Dengue virus infection in human cells by alpha, beta, and gamma interferons.

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Characterization of a highly attenuated Japanese encephalitis virus generated from molecularly cloned cDNA.

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Characterization of the E-138 (Glu/Lys) mutation in Japanese encephalitis virus by using a stable, full-length, infectious cDNA clone

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Common E protein determinants for attenuation of glycosaminoglycan-binding variants of Japanese encephalitis and West Nile viruses

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West Nile virus envelope protein inhibits dsRNA-induced innate immune responses

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A new strategy in design of +RNA virus infectious clones enabling their stable propagation in E. coli

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Development and Application of a Reverse Genetics System for Japanese Encephalitis Virus

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Robust production of infectious hepatitis C virus (HCV) from stably HCV cDNA-transfected human hepatoma cells

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10.1016/J.VACCINE.2009.03.007

7 January 2021

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DOI

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15 September 2017

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