Soluble decoy receptor 3 modulates the survival and formation of osteoclasts from multiple myeloma bone disease patients (Q39827231)

15 September 2017

Soluble decoy receptor 3 modulates the survival and formation of osteoclasts from multiple myeloma bone disease patients (English)

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9 July 2009

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https://scigraph.springernature.com/pub.10.1038/leu.2009.136

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T cells support osteoclastogenesis in an in vitro model derived from human periodontitis patients.

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TNF-alpha induces osteoclastogenesis by direct stimulation of macrophages exposed to permissive levels of RANK ligand

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Osteoclast differentiation independent of the TRANCE-RANK-TRAF6 axis

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Decoy receptor 3 (DcR3) induces osteoclast formation from monocyte/macrophage lineage precursor cells

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Attenuation of bone mass and increase of osteoclast formation in decoy receptor 3 transgenic mice

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Genomic amplification of a decoy receptor for Fas ligand in lung and colon cancer

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Overexpression of M68/DcR3 in human gastrointestinal tract tumors independent of gene amplification and its location in a four-gene cluster

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Amplification and expression of a decoy receptor for fas ligand (DcR3) in virus (EBV or HTLV-I) associated lymphomas

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A newly identified member of tumor necrosis factor receptor superfamily (TR6) suppresses LIGHT-mediated apoptosis

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TL1A is a TNF-like ligand for DR3 and TR6/DcR3 and functions as a T cell costimulator

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Ascites from epithelial ovarian cancer contain high levels of functional decoy receptor 3 (DcR3) and is associated with platinum resistance.

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A new mathematical model for relative quantification in real-time RT-PCR

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The death receptor DR5 is involved in TRAIL-mediated human osteoclast apoptosis

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Overexpression of decoy receptor 3 in hepatocellular carcinoma and its association with resistance to Fas ligand-mediated apoptosis

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Endogenous decoy receptor 3 blocks the growth inhibition signals mediated by Fas ligand in human pancreatic adenocarcinoma

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Two CD95 (APO-1/Fas) signaling pathways

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Immune evasion by tumours: involvement of the CD95 (APO-1/Fas) system and its clinical implications

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CC-chemokine ligand 20/macrophage inflammatory protein-3α and CC-chemokine receptor 6 are overexpressed in myeloma microenvironment related to osteolytic bone lesions.

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Multiple myeloma cells directly stimulate bone resorption in vitro by down-regulating mature osteoclast apoptosis

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T cells support osteoclastogenesis in an in vitro model derived from human multiple myeloma bone disease: the role of the OPG/TRAIL interaction

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Human myeloma cells stimulate the receptor activator of nuclear factor-kappa B ligand (RANKL) in T lymphocytes: a potential role in multiple myeloma bone disease

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7 January 2021

Peripheral blood T lymphocytes from patients with early rheumatoid arthritis express RANKL and interleukin-15 on the cell surface and promote osteoclastogenesis in autologous monocytes

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7 January 2021

Lymphocytes and synovial fluid fibroblasts support osteoclastogenesis through RANKL, TNFα, and IL-7 in anin vitromodel derived from human psoriatic arthritis

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7 January 2021

Mechanisms of spontaneous osteoclastogenesis in cancer with bone involvement

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7 January 2021

TNFalpha potently activates osteoclasts, through a direct action independent of and strongly synergistic with RANKL

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https://api.crossref.org/works/10.1038%2FLEU.2009.136

7 January 2021

Criteria for the classification of monoclonal gammopathies, multiple myeloma and related disorders: a report of the International Myeloma Working Group

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7 January 2021

Decoy receptor 3 expressed in rheumatoid synovial fibroblasts protects the cells against Fas-induced apoptosis

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7 January 2021

Apoptosis in rheumatoid arthritis--expression of Fas, Fas-L, p53, and Bcl-2 in rheumatoid synovial tissues

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7 January 2021

LIGHT (TNFSF14), a novel mediator of bone resorption, is elevated in rheumatoid arthritis

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https://api.crossref.org/works/10.1038%2FLEU.2009.136

7 January 2021

Identifiers

DOI

10.1038/LEU.2009.136

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Dimensions Publication ID

15 September 2017

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